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Different response of ENU-exposed and unexposed rat brain cells to cholera toxin at early passages in culture.

作者信息

Claisse P J, Roscoe J P

出版信息

Br J Cancer. 1981 Feb;43(2):240-4. doi: 10.1038/bjc.1981.36.

DOI:10.1038/bjc.1981.36
PMID:7470388
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2010508/
Abstract
摘要

相似文献

1
Different response of ENU-exposed and unexposed rat brain cells to cholera toxin at early passages in culture.在培养的早期传代中，经ENU处理和未经处理的大鼠脑细胞对霍乱毒素的不同反应。
Br J Cancer. 1981 Feb;43(2):240-4. doi: 10.1038/bjc.1981.36.
2
Neoplastic transformation of fetal rat brain cells in culture after exposure to ethylnitrosourea in vivo.体内暴露于乙基亚硝基脲后培养的胎鼠脑细胞的肿瘤转化。
J Natl Cancer Inst. 1975 Nov;55(5):1177-87. doi: 10.1093/jnci/55.5.1177.
3
Increased ability of ethylnitrosourea-exposed brain cells to survive suspension in agar.暴露于乙基亚硝基脲的脑细胞在琼脂中悬浮时存活能力增强。
Br J Cancer. 1980 Jun;41(6):992-5. doi: 10.1038/bjc.1980.180.
4
In vitro transformation of fetal brain cells from CDF rats exposed in utero to N-ethyl-N-nitrosourea: morphologic and immunologic studies.子宫内暴露于N-乙基-N-亚硝基脲的CDF大鼠胎脑细胞的体外转化：形态学和免疫学研究
J Natl Cancer Inst. 1980 May;64(5):1231-9.
5
Immunohistochemical detection of O6-ethyldeoxyguanosine in the rat brain after in vivo applications of N-ethyl-N-nitrosourea.体内应用N-乙基-N-亚硝基脲后大鼠脑内O6-乙基脱氧鸟苷的免疫组织化学检测
Cancer Res. 1983 Jun;43(6):2935-41.
6
Phenotypic properties of neoplastic cell lines developed from fetal rat brain cells in culture after exposure to ethylnitrosourea in vivo.
Z Krebsforsch Klin Onkol Cancer Res Clin Oncol. 1977 Aug 15;89(3):273-95. doi: 10.1007/BF00283783.
7
[Effects of ethylnitrosourea on the developing rat retina].[乙基亚硝基脲对发育中大鼠视网膜的影响]
Nippon Ganka Gakkai Zasshi. 1982;86(9):1224-35.
8
[Effect of tumor promoting agents on the in vitro malignant transformation of the fetal rat brain cells exposed in utero to ethylnitrosourea].
No To Shinkei. 1984 Feb;36(2):143-50.
9
The synthesis of brain chromosomal proteins after a pulse of the nervous system-specific carcinogen N-ethyl-N-nitrosourea to the fetal rat.在向胎鼠注射神经系统特异性致癌物N-乙基-N-亚硝基脲一次后，脑染色体蛋白的合成。
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10
Transplacental exposure to ethylnitrosourea and its effects on body, brain, and cerebellar development in the rat.经胎盘暴露于乙基亚硝基脲及其对大鼠身体、大脑和小脑发育的影响。
Cancer Res. 1978 May;38(5):1263-8.

本文引用的文献

1
Increased ability of ethylnitrosourea-exposed brain cells to survive suspension in agar.暴露于乙基亚硝基脲的脑细胞在琼脂中悬浮时存活能力增强。
Br J Cancer. 1980 Jun;41(6):992-5. doi: 10.1038/bjc.1980.180.
2
The fine structure of cloned cells from normal adult rat brain.来自正常成年大鼠大脑的克隆细胞的精细结构。
Experientia. 1980 Feb 15;36(2):194-6. doi: 10.1007/BF01953727.
3
Effect of 12-O-tetradecanoylphorbol-13-acetate on two charateristics of transformation acquired sequentially by ENU-exposed rat brain cells.12-O-十四酰佛波醇-13-乙酸酯对经ENU处理的大鼠脑细胞依次获得的两种转化特征的影响。
Br J Cancer. 1980 Nov;42(5):756-64. doi: 10.1038/bjc.1980.309.
4
In-vivo-in-vitro analysis of ethylnitrosourea-induced brain carcinogenesis in the rat.大鼠中乙基亚硝基脲诱导脑癌发生的体内-体外分析
Br Med Bull. 1980 Jan;36(1):33-8. doi: 10.1093/oxfordjournals.bmb.a071610.
5
Cholera toxin and analogues of cyclic AMP stimulate the growth of cultured human mammary epithelial cells.霍乱毒素和环磷酸腺苷类似物可刺激培养的人乳腺上皮细胞生长。
J Cell Physiol. 1980 Mar;102(3):317-21. doi: 10.1002/jcp.1041020306.
6
In vitro transformation of normal cells to tumor cells by carcinogenic hydrocarbons.致癌碳氢化合物使正常细胞在体外转化为肿瘤细胞。
J Natl Cancer Inst. 1965 Oct;35(4):641-61.
7
Mitomycin C treated cells as feeders.用丝裂霉素C处理过的细胞作为饲养层细胞。
Exp Cell Res. 1971 Nov;69(1):240-1. doi: 10.1016/0014-4827(71)90335-1.
8
Early appearance of "transformed" cells from the kidneys of rats treated with a "single" carcinogenic dose of dimethylnitrosamine (DMN) detected by culture in vitro.
Eur J Cancer (1965). 1974 Mar;10(3):177-84. doi: 10.1016/0014-2964(74)90151-0.
9
Studies of the morphology and tumorigenicity of experimental brain tumours in tissue culture.组织培养中实验性脑肿瘤的形态学与致瘤性研究。
Br J Exp Pathol. 1976 Feb;57(1):95-104.
10
A sequential in vivo-in vitro study of carcinogenesis induced in the rat brain by ethylnitrosourea.一项关于乙基亚硝基脲诱导大鼠脑内致癌作用的体内-体外序贯研究。
Nature. 1976 Jul 22;262(5566):314-6. doi: 10.1038/262314a0.