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Lipopolysaccharide binding protein: its role and therapeutical potential in inflammation and sepsis.

作者信息

Schumann R R, Lamping N, Kirschning C, Knopf H P, Hoess A, Herrmann F

机构信息

Max-Delbrück-Centrum für Molekulare Medizin (MDC), Berlin, Germany.

出版信息

Biochem Soc Trans. 1994 Feb;22(1):80-2. doi: 10.1042/bst0220080.

DOI:10.1042/bst0220080
PMID:7515836
Abstract
摘要

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Expansion of the Bactericidal/Permeability Increasing-like (BPI-like) protein locus in cattle.牛中杀菌/通透性增加样（BPI样）蛋白基因座的扩增。
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Serial monitoring of interleukin-1beta, soluble interleukin-2 receptor and lipopolysaccharide binding protein levels after death A comparative evaluation of potential postmortem markers of sepsis.死亡后白细胞介素-1β、可溶性白细胞介素-2受体和脂多糖结合蛋白水平的连续监测：脓毒症潜在死后标志物的比较评估
Int J Legal Med. 2005 Mar;119(2):80-7. doi: 10.1007/s00414-004-0481-0. Epub 2004 Sep 4.
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Receptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.
参与细菌性败血症和脓毒性休克的受体、介质及机制。
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Lipopolyamines: novel antiendotoxin compounds that reduce mortality in experimental sepsis caused by gram-negative bacteria.脂多胺：可降低革兰氏阴性菌所致实验性脓毒症死亡率的新型抗内毒素化合物。
Antimicrob Agents Chemother. 1999 Apr;43(4):912-9. doi: 10.1128/AAC.43.4.912.
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The lipopolysaccharide-binding protein is a secretory class 1 acute-phase protein whose gene is transcriptionally activated by APRF/STAT/3 and other cytokine-inducible nuclear proteins.脂多糖结合蛋白是一种分泌型1类急性期蛋白，其基因由APRF/STAT/3和其他细胞因子诱导的核蛋白转录激活。
Mol Cell Biol. 1996 Jul;16(7):3490-503. doi: 10.1128/MCB.16.7.3490.
6
Is sepsis a mediator-inhibitor mismatch?脓毒症是一种介质-抑制剂不匹配的情况吗？
Intensive Care Med. 1995 Nov;21 Suppl 2:S250-7. doi: 10.1007/BF01740763.
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The role of CD14 and lipopolysaccharide-binding protein (LBP) in the activation of different cell types by endotoxin.CD14和脂多糖结合蛋白（LBP）在内毒素激活不同细胞类型中的作用。
Med Microbiol Immunol. 1994 Dec;183(6):279-97. doi: 10.1007/BF00196679.