Granulocyte-colony stimulating factor (G-CSF) production of human fibroblasts (KMST-6/RAS line) transformed with 60Co gamma rays and c-Ha-ras oncogene.

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作者信息

Kawashima K, Mori M, Furusako S, Usuki H, Shimizu N, Namba M

出版信息

In Vitro Cell Dev Biol Anim. 1994 Apr;30A(4):199-201. doi: 10.1007/BF02632040.

DOI:10.1007/BF02632040

PMID:7520808

Abstract

摘要

相似文献

Granulocyte-colony stimulating factor (G-CSF) production of human fibroblasts (KMST-6/RAS line) transformed with 60Co gamma rays and c-Ha-ras oncogene.

In Vitro Cell Dev Biol Anim. 1994 Apr;30A(4):199-201. doi: 10.1007/BF02632040.

Human fibroblasts (KMST-6/RAS line) transformed with 60Co gamma-rays and c-Ha-ras oncogene constitutively produce a large amount of human granulocyte-colony stimulating factor (G-CSF).

Hum Cell. 1994 Jun;7(2):88-94.

Human fibroblasts (KMST-6/RAS cell line) transformed with 60Co gamma-rays and c-Ha-ras oncogene produce a large amount of granulocyte colony-stimulating factor (G-CSF); production is enhanced by cAMP, theophylline, and butyrate.

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Neoplastic transformation and characterization of human fibroblasts by treatment with 60Co gamma rays and the human c-Ha-ras oncogene.用60Coγ射线和人c-Ha-ras癌基因处理对人成纤维细胞进行肿瘤转化及特性研究

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Multistep carcinogenesis of normal human fibroblasts. Human fibroblasts immortalized by repeated treatment with Co-60 gamma rays were transformed into tumorigenic cells with Ha-ras oncogenes.正常人成纤维细胞的多步骤致癌作用。经钴-60γ射线反复处理永生化的人成纤维细胞被Ha-ras癌基因转化为致瘤细胞。

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Multi-step neoplastic transformation of normal human fibroblasts by Co-60 gamma rays and Ha-ras oncogenes.

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Neoplastic transformation of human diploid fibroblasts (KMST-6) by treatment with 60Co gamma rays.

Int J Cancer. 1985 Feb 15;35(2):275-80. doi: 10.1002/ijc.2910350221.

Malignant transformation of human fibroblasts previously immortalized with 60Co gamma rays.先前经60钴γ射线永生化的人成纤维细胞的恶性转化。

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Macrophage-colony stimulating factor is required for the production of neutrophil-promoting activity by mouse embryo fibroblasts deficient in G-CSF and GM-CSF.巨噬细胞集落刺激因子是G-CSF和GM-CSF缺陷的小鼠胚胎成纤维细胞产生促进中性粒细胞活性所必需的。

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本文引用的文献

Immortalization of normal human fibroblasts by treatment with 4-nitroquinoline 1-oxide.用4-硝基喹啉1-氧化物处理使正常人成纤维细胞永生化。

Int J Cancer. 1993 Feb 1;53(3):451-6. doi: 10.1002/ijc.2910530317.

In Vitro Cell Dev Biol Anim. 1993 Oct;29A(10):763-7. doi: 10.1007/BF02634342.

Long-term cultivation of a human colony-stimulating factor-producing cell line in a protein-free chemically defined medium.在无蛋白化学限定培养基中长期培养人集落刺激因子产生细胞系。

Cancer Res. 1984 Oct;44(10):4503-6.

Neoplastic transformation of human diploid fibroblasts (KMST-6) by treatment with 60Co gamma rays.

Int J Cancer. 1985 Feb 15;35(2):275-80. doi: 10.1002/ijc.2910350221.

Additive effects of interleukin 1 and tumour necrosis factor-alpha on the accumulation of the three granulocyte and macrophage colony-stimulating factor mRNAs in human endothelial cells.白细胞介素1和肿瘤坏死因子-α对人内皮细胞中三种粒细胞和巨噬细胞集落刺激因子mRNA积累的相加作用。

EMBO J. 1987 Aug;6(8):2261-5. doi: 10.1002/j.1460-2075.1987.tb02499.x.

Interleukin 1 stimulates fibroblasts to produce granulocyte-macrophage colony-stimulating activity and prostaglandin E2.白细胞介素1刺激成纤维细胞产生粒细胞-巨噬细胞集落刺激活性和前列腺素E2。

J Clin Invest. 1986 Jun;77(6):1857-63. doi: 10.1172/JCI112512.

Expression of colony-stimulating factor genes by normal human mesothelial cells and human malignant mesothelioma cells lines in vitro.

Blood. 1989 Aug 15;74(3):940-6.

Transcriptional and posttranscriptional modulation of myeloid colony-stimulating factor expression by tumor necrosis factor and other agents.肿瘤坏死因子及其他因子对髓系集落刺激因子表达的转录和转录后调控

Mol Cell Biol. 1988 Aug;8(8):3432-8. doi: 10.1128/mcb.8.8.3432-3438.1988.

Human fibroblasts produce granulocyte-CSF, macrophage-CSF, and granulocyte-macrophage-CSF following stimulation by interleukin-1 and poly(rI).poly(rC).人成纤维细胞在受到白细胞介素 -1 和聚肌苷酸 - 聚胞苷酸刺激后会产生粒细胞集落刺激因子、巨噬细胞集落刺激因子以及粒细胞 - 巨噬细胞集落刺激因子。

Blood. 1988 Sep;72(3):860-6.

Analysis of abnormal expression of g-csf gene in a novel tumor cell line (KHC 287) elaborating G-CSF, IL-1 and IL-6 with co-amplification of c-myc and c-ki-ras.

Int J Cancer. 1991 May 30;48(3):428-33. doi: 10.1002/ijc.2910480321.

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