The CD28/CTLA-4:B7 receptor system in experimental autoimmune encephalomyelitis.
作者信息
Linsley P S
出版信息
J Clin Invest. 1995 Jun;95(6):2429-30. doi: 10.1172/JCI117940.
Abstract
摘要
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Immunotherapy of autoimmune disease.自身免疫性疾病的免疫疗法。
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Long-term acceptance of major histocompatibility complex mismatched cardiac allografts induced by CTLA4Ig plus donor-specific transfusion.CTLA4Ig联合供体特异性输血诱导主要组织相容性复合体不匹配心脏同种异体移植物的长期接受
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Partial T cell signaling: altered phospho-zeta and lack of zap70 recruitment in APL-induced T cell anergy.部分T细胞信号传导：急性早幼粒细胞白血病诱导的T细胞无反应性中磷酸化ζ链改变及ZAP70募集缺失
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Genes for SMA: multum in parvo.脊髓性肌萎缩症的基因：麻雀虽小，五脏俱全。
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6
Role of B7:CD28/CTLA-4 in the induction of chronic relapsing experimental allergic encephalomyelitis.B7:CD28/CTLA-4在慢性复发性实验性自身免疫性脑脊髓炎诱导中的作用。
J Immunol. 1995 Feb 1;154(3):1481-90.
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The B7 and CD28 receptor families.B7和CD28受体家族。
Immunol Today. 1994 Jul;15(7):321-31. doi: 10.1016/0167-5699(94)90080-9.
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Treatment of murine lupus with CTLA4Ig.用CTLA4Ig治疗小鼠狼疮。
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CTLA-4 is a second receptor for the B cell activation antigen B7.细胞毒性T淋巴细胞相关抗原4是B细胞活化抗原B7的第二种受体。
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Science. 1992 Aug 7;257(5071):792-5. doi: 10.1126/science.1496399.
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