Eberhard P, Manueddu C, Hoffmeyer P, Vasey H
Service de Chirurige Orthopédique et Traumatologique, Hôpital communal, La Chaux-de-Fonds.
Rev Chir Orthop Reparatrice Appar Mot. 1995;81(1):78-80.
Progressive dissolution of the supero-lateral fragment of a bipartite patella is reported.
The patient, a 44 year old professor of gymnastics, was examined for long standing anterior knee pain. For many years, his daily activities included forceful knee flexion exercises. Serial radiographs, between 1986 and 1990, revealed a progressive fragmentation of the accessory ossification center and accompanying calcific deposits in the lateral patellar retinaculum.
Surgical excision of the calcific mass and accessory ossification center relieved the painful symptomatology completely, allowing the patient full normal function at one year's follow-up.
The pathological findings were compatible with a dystrophic calcinosis associated with detritic synovitis. A pathogenic mechanism is hypothesized for this unusual and progressive course of a common condition: Repetitive trauma, in this case forceful knee flexion exercises, shear microscopic bony fragments of the poorly vascularized accessory ossification center. These microscopic fragments act as deposits of an apatite crystal which go on to induce the secondary soft tissue calcifications seen in our patient.
报告二分髌骨上外侧碎片的进行性溶解。
患者为一名44岁的体操教授,因长期膝关节前部疼痛接受检查。多年来,他的日常活动包括强力屈膝练习。1986年至1990年间的系列X线片显示,附属骨化中心逐渐碎裂,并伴有髌外侧支持带钙化沉积。
手术切除钙化肿块和附属骨化中心后,疼痛症状完全缓解,患者在一年随访时功能完全恢复正常。
病理结果与碎屑性滑膜炎相关的营养不良性钙化症相符。针对这种常见疾病的不寻常且进行性病程提出了一种致病机制假说:反复创伤,在本病例中为强力屈膝练习,导致血管化不良的附属骨化中心产生微小骨折碎片。这些微小碎片充当磷灰石晶体的沉积物,进而引发了我们患者所见的继发性软组织钙化。