The role of H(+)-ATPases in urinary acidification.

Suppr

超能文献

作者信息

Constantinescu A

机构信息

Albert Einstein College of Medicine, Department of Pediatrics, Bronx, NY 10461, USA.

出版信息

Pediatr Nephrol. 1995 Aug;9(4):510-3. doi: 10.1007/BF00866742.

DOI:10.1007/BF00866742

PMID:7577421

Abstract

摘要

相似文献

The role of H(+)-ATPases in urinary acidification.

Pediatr Nephrol. 1995 Aug;9(4):510-3. doi: 10.1007/BF00866742.

The enzymatic correlate for the renal adaptation to chronic metabolic acidosis and metabolic alkalosis in rat nephron.大鼠肾单位对慢性代谢性酸中毒和代谢性碱中毒的肾脏适应性的酶学关联。

Trans Assoc Am Physicians. 1988;101:264-9.

Experimental studies in distal urinary acidification: bringing the bedside to the bench.远端尿液酸化的实验研究：将床边研究引入实验室。

Semin Nephrol. 1999 Mar;19(2):188-94.

Decreased activity of the mitochondrial ATP-synthase in fibroblasts from children with late-infantile and juvenile neuronal ceroid lipofuscinosis.

J Inherit Metab Dis. 1996;19(2):130-2. doi: 10.1007/BF01799411.

The effect of metabolic acidosis and alkalosis on the H+-ATPase of rat cerebral microvessels.代谢性酸中毒和碱中毒对大鼠脑微血管H⁺-ATP酶的影响。

Life Sci. 1997;61(22):2247-53. doi: 10.1016/s0024-3205(97)00927-2.

Regulation of the mitochondrial ATP-synthase in skeletal muscle from children--a new diagnostic tool.

J Inherit Metab Dis. 1996;19(2):137-9. doi: 10.1007/BF01799413.

Changes in H-ATPase activity in the distal nephron segments of the rat during metabolic acidosis and alkalosis.代谢性酸中毒和碱中毒期间大鼠远端肾单位节段中H-ATP酶活性的变化。

Contrib Nephrol. 1991;92:39-45. doi: 10.1159/000420075.

Mutations in the gene encoding B1 subunit of H+-ATPase cause renal tubular acidosis with sensorineural deafness.编码H⁺-ATP酶B1亚基的基因突变会导致伴有感音神经性耳聋的肾小管性酸中毒。

Nat Genet. 1999 Jan;21(1):84-90. doi: 10.1038/5022.

Mutations in ATP6N1B, encoding a new kidney vacuolar proton pump 116-kD subunit, cause recessive distal renal tubular acidosis with preserved hearing.编码一种新的肾空泡质子泵116-kD亚基的ATP6N1B基因突变导致隐性远端肾小管酸中毒且听力保留。

Nat Genet. 2000 Sep;26(1):71-5. doi: 10.1038/79208.

The connecting tubule is the main site of the furosemide-induced urinary acidification by the vacuolar H+-ATPase.连接小管是速尿通过液泡H⁺-ATP酶诱导尿液酸化的主要部位。

Kidney Int. 2006 Nov;70(10):1706-16. doi: 10.1038/sj.ki.5001851. Epub 2006 Sep 20.

本文引用的文献

Identification of two subunit A isoforms of the vacuolar H(+)-ATPase in human osteoclastoma.人骨巨细胞瘤中液泡H(+) -ATP酶两种A亚基同工型的鉴定

J Biol Chem. 1993 Apr 5;268(10):7075-80.

[Classification of renal tubular acidosis. Recent data].[肾小管性酸中毒的分类。最新数据]

Ann Pediatr (Paris). 1993 Feb;40(2):81-9.

Functional characterization of three intercalated cell subtypes in the rabbit outer cortical collecting duct.兔肾外皮质集合管中三种闰细胞亚型的功能特性

J Clin Invest. 1994 Jan;93(1):417-23. doi: 10.1172/JCI116976.

Mechanisms of urinary K+ and H+ excretion: primary structure and functional expression of a novel H,K-ATPase.尿钾离子和氢离子排泄机制：一种新型氢钾ATP酶的一级结构与功能表达

J Cell Biol. 1993 Dec;123(6 Pt 1):1421-9. doi: 10.1083/jcb.123.6.1421.

Cloning and tissue distribution of subunits C, D, and E of the human vacuolar H(+)-ATPase.

Biochem Biophys Res Commun. 1993 Nov 30;197(1):15-21. doi: 10.1006/bbrc.1993.2434.

Primary sequence and functional expression of a novel beta subunit of the P-ATPase gene family.P-ATP 酶基因家族一种新型β亚基的一级序列与功能表达

Pflugers Arch. 1993 Dec;425(5-6):446-52. doi: 10.1007/BF00374871.

The E subunit of vacuolar H(+)-ATPase localizes close to the centromere on human chromosome 22.

Hum Mol Genet. 1994 Feb;3(2):335-9. doi: 10.1093/hmg/3.2.335.

Heterogeneity of vacuolar H(+)-ATPase: differential expression of two human subunit B isoforms.液泡H(+) -ATP酶的异质性：两种人类亚基B同工型的差异表达

Biochem J. 1994 Oct 1;303 ( Pt 1)(Pt 1):191-8. doi: 10.1042/bj3030191.

Mechanisms of active H+ secretion in the proximal tubule.近端小管中活性H⁺分泌的机制。

Am J Physiol. 1983 Dec;245(6):F647-59. doi: 10.1152/ajprenal.1983.245.6.F647.

Microperfusion study of distal tubular potassium and sodium transfer in rat kidney.大鼠肾脏远曲小管钾和钠转运的微量灌注研究。

Am J Physiol. 1966 Sep;211(3):548-59. doi: 10.1152/ajplegacy.1966.211.3.548.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验