肌动蛋白细胞骨架。细胞内细菌运动的缺失环节？
Actin cytoskeleton. Missing link for intracellular bacterial motility?
作者信息
Pollard T D
机构信息
Department of Cell Biology and Anatomy, Johns Hopkins Medical School, Baltimore, Maryland 21205, USA.
出版信息
Curr Biol. 1995 Aug 1;5(8):837-40. doi: 10.1016/s0960-9822(95)00167-9.
PMID:7583135
Abstract
Vasodilator-stimulated phosphoprotein (VASP) associates with virulence factors on the surface of intracellular bacteria; by binding to profilin, VASP may help direct the actin assembly that appears to drive bacterial motility.
摘要
血管舒张刺激磷蛋白（VASP）与细胞内细菌表面的毒力因子相关联；通过与丝切蛋白结合，VASP可能有助于引导肌动蛋白组装，而肌动蛋白组装似乎驱动细菌运动。
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