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Listeria and Shigella actin-based motility in host cells.

作者信息

Southwick F S, Purich D L

机构信息

Department of Medicine, University of Florida College of Medicine, Gainesville 32610, USA.

出版信息

Trans Am Clin Climatol Assoc. 1998;109:160-72; discussion 172-3.

Abstract
摘要

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本文引用的文献

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Vinculin proteolysis unmasks an ActA homolog for actin-based Shigella motility.
J Cell Biol. 1997 Sep 22;138(6):1255-64. doi: 10.1083/jcb.138.6.1255.
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Functional analysis of the Shigella flexneri IpaC invasin by insertional mutagenesis.
Infect Immun. 1997 May;65(5):1599-605. doi: 10.1128/iai.65.5.1599-1605.1997.
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Intracellular pathogenesis of listeriosis.
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The secreted Ipa complex of Shigella flexneri promotes entry into mammalian cells.
Proc Natl Acad Sci U S A. 1996 Feb 6;93(3):1254-8. doi: 10.1073/pnas.93.3.1254.
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How profilin promotes actin filament assembly in the presence of thymosin beta 4.
Cell. 1993 Dec 3;75(5):1007-14. doi: 10.1016/0092-8674(93)90544-z.
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Arrest of Listeria movement in host cells by a bacterial ActA analogue: implications for actin-based motility.
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