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一项针对15名腭裂晚期修复治疗的唇腭裂儿童言语情况的纵向研究。

A longitudinal study of speech in 15 children with cleft lip and palate treated by late repair of the hard palate.

作者信息

Lohmander-Agerskov A, Söderpalm E, Friede H, Lilja J

机构信息

Department of Logopedics and Phoniatrics, Göteborg University, Sweden.

出版信息

Scand J Plast Reconstr Surg Hand Surg. 1995 Mar;29(1):21-31. doi: 10.3109/02844319509048419.

DOI:10.3109/02844319509048419
PMID:7597386
Abstract

Since 1975, children with cleft lip and palate living in the western part of Sweden have been treated according to a regimen of early repair of the soft palate (at the age of 6-8 months) and late hard palate closure (at about 8-9 years of age). The present paper is a longitudinal study of 15 consecutive patients whose speech development was analysed at the mean ages (years:months) of 5:3, 7:0, 8:5, and 9:7 years. Hypernasality gradually decreased over the years whereas nasal escape almost completely ceased after closure of the residual cleft. There was no glottal articulation at any age. Despite the fact that retraction of apicodental consonants decreased in frequency with age and presumably with speech therapy, it was the main problem throughout the observation period. It was presumably caused by the residual cleft in the hard palate compensating for subnormal pressure in front of the opening to the nasal cavity.

摘要

自1975年以来,瑞典西部唇腭裂患儿一直按照软腭早期修复(6至8个月龄)和硬腭晚期闭合(约8至9岁)的方案进行治疗。本文是一项对15例连续患者的纵向研究,分析了他们在平均年龄(岁:月)5:3、7:0、8:5和9:7岁时的言语发展情况。多年来,高鼻音逐渐减轻,而在残余腭裂闭合后,鼻漏气几乎完全停止。在任何年龄都没有声门发音。尽管齿龈塞音的后缩频率随着年龄增长以及可能随着言语治疗而降低,但在整个观察期内,这都是主要问题。这可能是由于硬腭的残余腭裂补偿了鼻腔开口前方的压力不足所致。

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引用本文的文献

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Three-dimensional analysis of the deciduous dentition of patients with bilateral cleft lip and palate and delayed cleft closure.双侧唇腭裂及腭裂延迟闭合患者乳牙列的三维分析
J Orofac Orthop. 2009 May;70(3):237-46. doi: 10.1007/s00056-009-8814-8. Epub 2009 May 31.
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Development of the residual cleft in the hard palate after velar repair in a 2-stage palatal repair regimen.
两阶段腭裂修复方案中腭帆修复后硬腭残留裂隙的发展情况。
J Orofac Orthop. 1998;59(5):286-300. doi: 10.1007/BF01321795.