Giordano C P, Scott D, Koval K J, Kummer F, Atik T, Desai P
Hospital for Joint Diseases, Orthopaedic Institute, New York, New York 10003, USA.
J Trauma. 1995 Jun;38(6):907-9. doi: 10.1097/00005373-199506000-00014.
A biomechanical study was performed to examine a proposed mechanism of fracture blister formation. Sixty cadaver ankle skin specimens were subjected to several levels of uniaxial strain and examined histologically. Dermal-epidermal separation patterns similar to those found histologically in previous studies of biopsied fracture blisters were seen in specimens strained 152% and greater. These findings support the hypothesis that fracture blisters can result from strain developed in the skin during initial fracture deformation.
进行了一项生物力学研究，以检验一种关于骨折水疱形成的推测机制。对60个尸体踝关节皮肤标本施加了几个水平的单轴应变，并进行了组织学检查。在应变达到152%及更高的标本中，观察到了与先前活检骨折水疱的组织学研究中发现的类似的真皮-表皮分离模式。这些发现支持了这样一种假说，即骨折水疱可能是由初始骨折变形过程中皮肤产生的应变导致的。
