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[抗休克服对创伤性休克伤员抗低血压作用机制的研究]

[Study of mechanisms of the antihypotensive effects of antishock suits in injured with traumatic shock].

作者信息

Pikovskiĭ V Iu, Storozhenko I N, Sud'in V I, Artamoshina M P, Petrov O V

出版信息

Anesteziol Reanimatol. 1995 Jan-Feb(1):22-5.

PMID:7605028
Abstract

The mechanisms of antihypotensive action of antishock overalls (ASO) were studied on the basis of comparison of 30 patients with traumatic shock at the prehospital stage of medical care; in 15 of these ASO was used, in the other 15 traditional antishock intensive care. Use of a portable device for noninvasive measurement of cardiac stroke volume at the prehospital stage permitted us detect the principal mechanisms of liquidation of shock-induced arterial hypotension with the use of ASO: increase of cardiac output at the expense of autohemotransfusion from the lower part of the body to the upper vital organs with increase in the volume of venous blood restitution to the heart, the increase of total peripheral vascular resistance being negligible. Use of ASO in a complex of intensive care measures for the treatment of traumatic shock at the prehospital stage is physiological and conducive to optimization of medical care rendered to victims at the early stages of treatment.

摘要

基于对30例处于院前医疗阶段的创伤性休克患者的比较,研究了抗休克工作服(ASO)的抗低血压作用机制;其中15例使用了ASO,另外15例采用传统的抗休克重症监护。在院前阶段使用便携式无创心输出量测量装置,使我们能够检测出使用ASO消除休克引起的动脉低血压的主要机制:通过自体血液从身体下部向重要的上部器官转移,增加心输出量,同时增加静脉血回流到心脏的量,而总外周血管阻力的增加可忽略不计。在院前阶段将ASO用于创伤性休克治疗的重症监护综合措施中,具有生理学意义,有助于优化对受害者在治疗早期提供的医疗护理。

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