A clonal deletion model for Ir gene control of the immune response.
作者信息
Schwartz R H
出版信息
Scand J Immunol. 1978;7(1):3-10. doi: 10.1111/j.1365-3083.1978.tb00420.x.
Abstract
摘要
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A clonal deletion model for Ir gene control of the immune response.一种用于免疫应答Ir基因控制的克隆缺失模型。
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The immune response genes of the major histocompatibility complex.主要组织相容性复合体的免疫反应基因。
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Immune response (Ir) genes expressed at macrophage-B lymphocyte interactions.在巨噬细胞与B淋巴细胞相互作用时表达的免疫反应（Ir）基因。
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Regulation of immune responses: analysis with lymphocyte clones.免疫反应的调节：淋巴细胞克隆分析
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[Regulation of immune system functions].[免疫系统功能的调节]
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Monoclonal T cell responses to two epitopes on a single immunogen controlled by two distinct genes.单克隆T细胞对由两个不同基因控制的单一免疫原上的两个表位产生反应。
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Mhc restriction and Ir genes.主要组织相容性复合体限制与免疫应答基因
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T-B reciprocity. An Ia-restricted epitope-specific circuit regulating T cell-B cell interaction and antibody specificity.T细胞与B细胞的相互作用。一种受Ia限制的表位特异性回路，调节T细胞与B细胞的相互作用及抗体特异性。
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Absence of Ir gene control of T cells recognizing foreign antigen in the context of allogenic MHC molecules.在同种异体MHC分子的背景下，不存在Ir基因对识别外来抗原的T细胞的控制。
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Non-H-2 and H-2-linked immune response genes control the cytotoxic T-cell response to H-Y.非H-2和H-2连锁免疫反应基因控制细胞毒性T细胞对H-Y的反应。
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Antigen-reactive T clones. III. Low responder antigen-presenting cells function effectively to present antigen to selected T cell clones derived from (High Responder x Low Responder)F1 mice.抗原反应性T克隆。III.低反应性抗原呈递细胞能有效地将抗原呈递给源自（高反应性×低反应性）F1小鼠的选定T细胞克隆。
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Molecular events in the processing of avidin by antigen-presenting cells (APC). III. Activation of T-lymphocyte lines and H-2 restriction are mediated by processed avidin associated with I-region gene products.抗原呈递细胞（APC）处理抗生物素蛋白过程中的分子事件。III. 与I区基因产物相关的经处理抗生物素蛋白介导T淋巴细胞系的激活和H-2限制性。
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H-2 effects on cell-cell interactions in the response to single non-H-2 alloantigens. VI. H-2Kb mutants differentially regulate the immune response to the H-4.2 alloantigen.H-2对针对单一非H-2同种异体抗原反应中细胞间相互作用的影响。VI. H-2Kb突变体对H-4.2同种异体抗原免疫反应的差异调节。
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Mechanisms of genetic control of immune responses. I. Evidence for distinct multi-step helper T-cell pathways in cellular and humoral responses to GAT.免疫反应的遗传控制机制。I. 对谷氨酸-丙氨酸-酪氨酸（GAT）的细胞免疫和体液免疫反应中不同多步骤辅助性T细胞途径的证据。
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Neonatal tolerance of major histocompatibility complex antigens alters Ir gene control of the cytotoxic T cell response to vaccinia virus.主要组织相容性复合体抗原的新生儿耐受性改变了Ir基因对细胞毒性T细胞针对痘苗病毒反应的控制。
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