Trick G L, Steinman S B, Amyot M
School of Optometry, University of Montreal, Quebec, Canada.
Vision Res. 1995 Aug;35(15):2225-33. doi: 10.1016/0042-6989(94)00311-4.
The mechanisms mediating impaired motion perception in glaucoma were investigated. Direction discrimination thresholds for low (4.2 deg/sec) and high (12.5 deg/sec) velocity random-dot kinematograms were measured in controls and patients with glaucoma or ocular hypertension. Thresholds were elevated significantly in glaucoma patients and individual ocular hypertensives. Threshold elevations were not due to blur or pupil size. After compensating for motion reversals, high but not low velocity thresholds remained elevated. Only high velocity thresholds correlated with differential luminance sensitivity. A hypothesis that different mechanisms mediate glaucoma-induced deficits at high and low velocities is presented.
对青光眼患者运动感知受损的介导机制进行了研究。测量了对照组、青光眼患者或高眼压症患者对低速度（4.2度/秒）和高速度（12.5度/秒）随机点运动图的方向辨别阈值。青光眼患者和个别高眼压症患者的阈值显著升高。阈值升高并非由模糊或瞳孔大小所致。在对运动反转进行补偿后，高速度阈值仍升高，而低速度阈值未升高。只有高速度阈值与差异亮度敏感度相关。本文提出了一个假说，即不同机制介导了青光眼在高、低速度下所致的缺陷。
