丁香假单胞菌丁香致病变种的植物毒素可可逆性抑制质膜H(+) -ATP酶并破坏单层脂质体。
Pseudomonas syringae pv. syringae phytotoxins reversibly inhibit the plasma membrane H(+)-ATPase and disrupt unilamellar liposomes.
作者信息
Camoni L, Di Giorgio D, Marra M, Aducci P, Ballio A
机构信息
Dipartimento di Biologia, Università di Roma, Tor Vergata, Italy.
出版信息
Biochem Biophys Res Commun. 1995 Sep 5;214(1):118-24. doi: 10.1006/bbrc.1995.2264.
The Pseudomonas syringae pv. syringae phytotoxins syringomycin-E and syringopeptins 22-A and 25-A reversibly and noncompetitively inhibit purified H(+)-ATPase solubilized from plasma membrane of maize roots. Moreover, they increase the passive permeability to protons in phosphatidylcholine/phosphatidylethanolamine liposomes. Both effects are more pronounced with syringopeptins than with syringomycin-E. Activity on phospholipid bilayers is detectable at phytotoxin concentrations not affecting H(+)-ATPase activity.
丁香假单胞菌丁香致病变种的植物毒素丁香霉素 -E、丁香肽素22 -A和25 -A可逆且非竞争性地抑制从玉米根质膜中溶解出来的纯化H⁺-ATP酶。此外，它们还增加了质子在磷脂酰胆碱/磷脂酰乙醇胺脂质体中的被动通透性。丁香肽素的这两种效应比丁香霉素 -E更明显。在不影响H⁺-ATP酶活性的植物毒素浓度下就能检测到其对磷脂双层的作用。
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