脱氧核糖核酸修复途径中的突变对大肠杆菌K-12菌株对呋喃妥因敏感性的影响。
Effect of mutations in deoxyribonucleic acid repair pathways on the sensitivity of Escherichia coli K-12 strains to nitrofurantoin.
作者信息
Jenkins S T, Bennett P M
出版信息
J Bacteriol. 1976 Mar;125(3):1214-6. doi: 10.1128/jb.125.3.1214-1216.1976.
Abstract
Escherichia coli K-12 strains that carry mutations in one or more genes coding for proteins involved in repair of deoxyribonucleic acid lesions are more sensitive to the antibiotic nitrofurantoin than are the nonmutant parent strains.
摘要
在一个或多个参与脱氧核糖核酸损伤修复的蛋白质编码基因中携带突变的大肠杆菌K-12菌株，比未突变的亲本菌株对抗生素呋喃妥因更敏感。
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本文引用的文献
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Mode of action of nitrofurazone.呋喃西林的作用方式。
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