Gout: questions that still need to be answered.
作者信息
Terkeltaub R A
机构信息
Department of Medicine, VA Medical Center, San Diego, CA 92161, USA.
出版信息
Ann Rheum Dis. 1995 Feb;54(2):79-81. doi: 10.1136/ard.54.2.79.
Abstract
摘要
相似文献
1
Gout: questions that still need to be answered.痛风：仍需解答的问题。
Ann Rheum Dis. 1995 Feb;54(2):79-81. doi: 10.1136/ard.54.2.79.
2
[Animal model for gout].[痛风动物模型]
Jikken Dobutsu. 1982 Apr;31(2):143-51.
3
[Some problems of uric acid pathology].[尿酸病理学的一些问题]
Postepy Hig Med Dosw. 1976 Jul-Aug;30(4):553-9.
4
Urate-oxidase for the treatment of tophaceous gout in heart transplant recipients. A report of three cases.尿酸氧化酶治疗心脏移植受者痛风石性痛风。三例报告。
Rev Rhum Engl Ed. 1995 May;62(5):392-4.
5
Pathogenesis of gout.痛风的发病机制。
Ann Intern Med. 2005 Oct 4;143(7):499-516. doi: 10.7326/0003-4819-143-7-200510040-00009.
6
Deposition of urate crystals in man.人体中尿酸盐结晶的沉积。
Ann Rheum Dis. 1969 May;28(3):327. doi: 10.1136/ard.28.3.327-a.
7
A conserved role of the insulin-like signaling pathway in diet-dependent uric acid pathologies in Drosophila melanogaster.胰岛素样信号通路在果蝇依赖饮食的尿酸病理中的保守作用。
PLoS Genet. 2019 Aug 15;15(8):e1008318. doi: 10.1371/journal.pgen.1008318. eCollection 2019 Aug.
8
Physiologic and biochemical basis for the treatment of gout and hyperuricemia.痛风和高尿酸血症治疗的生理与生化基础
Med Clin North Am. 1969 Nov;53(6):1331-47.
9
[Physiology and dynamics of uric acid in hyperuricemia].[高尿酸血症中尿酸的生理与动态变化]
Nihon Rinsho. 2008 Apr;66(4):669-74.
10
[Gout due to disorder of urate binding proteins (the urate-binding alpha 1-2 globulin deficiency)].[尿酸盐结合蛋白紊乱（尿酸盐结合性α1-2球蛋白缺乏）所致痛风]
Ryoikibetsu Shokogun Shirizu. 1998(18 Pt 1):478-80.
本文引用的文献
1
The adhesion molecules of inflammation.炎症的黏附分子
Arthritis Rheum. 1993 Feb;36(2):147-57. doi: 10.1002/art.1780360204.
2
Colchicine, crystals, and neutrophil tyrosine phosphorylation.秋水仙碱、晶体与中性粒细胞酪氨酸磷酸化
J Clin Invest. 1993 Oct;92(4):1602-3. doi: 10.1172/JCI116742.
3
Crystal-induced neutrophil activation. III. Inflammatory microcrystals induce a distinct pattern of tyrosine phosphorylation in human neutrophils.晶体诱导的中性粒细胞活化。III. 炎性微晶在人中性粒细胞中诱导独特的酪氨酸磷酸化模式。
J Clin Invest. 1993 Apr;91(4):1649-55. doi: 10.1172/JCI116373.
4
Rapid acceleration of neutrophil apoptosis by tumor necrosis factor-alpha.肿瘤坏死因子-α导致中性粒细胞凋亡迅速加速。
Int Immunol. 1993 Jun;5(6):691-4. doi: 10.1093/intimm/5.6.691.
5
Gout without hyperuricemia.无高尿酸血症的痛风
JAMA. 1994 Jan 26;271(4):302-3.
6
Hyperuricemia and urate nephropathy in urate oxidase-deficient mice.尿酸氧化酶缺陷小鼠的高尿酸血症和尿酸盐肾病
Proc Natl Acad Sci U S A. 1994 Jan 18;91(2):742-6. doi: 10.1073/pnas.91.2.742.
7
Inhibition of crystal-induced neutrophil activation by a protein tyrosine kinase inhibitor.蛋白酪氨酸激酶抑制剂对晶体诱导的中性粒细胞活化的抑制作用
J Leukoc Biol. 1994 Jan;55(1):112-9. doi: 10.1002/jlb.55.1.112.
8
Oxidized LDL induces monocytic cell expression of interleukin-8, a chemokine with T-lymphocyte chemotactic activity.氧化型低密度脂蛋白可诱导单核细胞表达白细胞介素-8，这是一种具有T淋巴细胞趋化活性的趋化因子。
Arterioscler Thromb. 1994 Jan;14(1):47-53. doi: 10.1161/01.atv.14.1.47.
9
Protein tyrosine kinase activation is required for lipopolysaccharide induction of cytokines in human blood monocytes.蛋白酪氨酸激酶激活是脂多糖诱导人血单核细胞产生细胞因子所必需的。
J Immunol. 1993 Dec 15;151(12):6692-700.
10
Changes in the proteins coating monosodium urate crystals during active and subsiding inflammation. Immunogold studies of synovial fluid from patients with gout and of fluid obtained using the rat subcutaneous air pouch model.活动期和消退期炎症期间尿酸钠晶体表面蛋白质的变化。痛风患者滑液及大鼠皮下气囊模型所获液体的免疫金研究。
Arthritis Rheum. 1993 Sep;36(9):1274-85. doi: 10.1002/art.1780360912.
Zotero 插件