The response-to-retention hypothesis of early atherogenesis.
作者信息
Williams K J, Tabas I
机构信息
Division of Endocrinology and Metabolic Diseases, Thomas Jefferson University, Philadelphia, PA 19107-6799, USA.
出版信息
Arterioscler Thromb Vasc Biol. 1995 May;15(5):551-61. doi: 10.1161/01.atv.15.5.551.
Abstract
摘要
相似文献
1
The response-to-retention hypothesis of early atherogenesis.早期动脉粥样硬化的反应-留存假说。
Arterioscler Thromb Vasc Biol. 1995 May;15(5):551-61. doi: 10.1161/01.atv.15.5.551.
2
Oxidised low density lipoproteins and atherogenesis.氧化型低密度脂蛋白与动脉粥样硬化的发生
Br Heart J. 1993 Jun;69(6):476-8. doi: 10.1136/hrt.69.6.476.
3
Dietary polyunsaturated fat decreases interaction between low density lipoproteins and arterial proteoglycans.膳食多不饱和脂肪可减少低密度脂蛋白与动脉蛋白聚糖之间的相互作用。
Lipids. 1994 Sep;29(9):635-41. doi: 10.1007/BF02536098.
4
Role of extracellular retention of low density lipoproteins in atherosclerosis.低密度脂蛋白细胞外潴留在动脉粥样硬化中的作用。
Curr Opin Lipidol. 2000 Oct;11(5):451-6. doi: 10.1097/00041433-200010000-00002.
5
Atherosclerosis: the low-density lipoprotein receptor hypothesis.
Metabolism. 1977 Nov;26(11):1257-75. doi: 10.1016/0026-0495(77)90119-6.
6
[Lipoproteins and their metabolism].
Kardiol Pol. 1992;36(2):110-4.
7
Transfer of lipoproteins from plasma to the cell populations of the normal and atherosclerotic arterial tissue.
Eur Heart J. 1990 Aug;11 Suppl E:158-63. doi: 10.1093/eurheartj/11.suppl_e.158.
8
[Cellular and extracellular lipid binding].
Przegl Lek. 1988;45(7):588-91.
9
Mechanisms of glycation in atherogenesis.
Med Hypotheses. 1993 Mar;40(3):174-81. doi: 10.1016/0306-9877(93)90207-7.
10
Arterial metabolism of lipoproteins in relation to atherogenesis.脂蛋白的动脉代谢与动脉粥样硬化形成的关系。
Ann N Y Acad Sci. 1990;598:125-35. doi: 10.1111/j.1749-6632.1990.tb42284.x.
引用本文的文献
1
Divergence in Estimated Risks and Actual Rates of Atherosclerotic Cardiovascular Events and their Management in a Longitudinal Cohort of Patients with Dermatomyositis with Skin Involvement.皮肤受累的皮肌炎患者纵向队列中动脉粥样硬化性心血管事件的估计风险与实际发生率及其管理的差异
JID Innov. 2025 Jul 7;5(6):100399. doi: 10.1016/j.xjidi.2025.100399. eCollection 2025 Nov.
2
The Role of Lysyl Oxidase in the Pathological Stage of Atherosclerosis: Structural Stabilizer or Disease Driver?赖氨酰氧化酶在动脉粥样硬化病理阶段的作用：结构稳定剂还是疾病驱动因素？
Curr Atheroscler Rep. 2025 Jul 5;27(1):69. doi: 10.1007/s11883-025-01312-z.
3
Atherosclerosis: from lipid-lowering and anti-inflammatory therapies to targeting arterial retention of ApoB-containing lipoproteins.动脉粥样硬化：从降脂和抗炎疗法到靶向含载脂蛋白B脂蛋白的动脉潴留
Front Immunol. 2025 Jun 9;16:1485801. doi: 10.3389/fimmu.2025.1485801. eCollection 2025.
4
Beyond cholesterol: linking the conformation of apolipoprotein B to atherogenesis.超越胆固醇：载脂蛋白B的构象与动脉粥样硬化的关联
Curr Opin Lipidol. 2025 Oct 1;36(5):226-231. doi: 10.1097/MOL.0000000000000997. Epub 2025 Jun 23.
5
Bruch's membrane heparan sulfate retains lipoproteins in the early stages of age-related macular degeneration.布鲁赫膜硫酸乙酰肝素在年龄相关性黄斑变性的早期阶段保留脂蛋白。
Proc Natl Acad Sci U S A. 2025 Jun 17;122(24):e2500727122. doi: 10.1073/pnas.2500727122. Epub 2025 Jun 13.
6
Modern therapy of patients with lupus erythematosus must include appropriate management of their heightened rates of atherosclerotic cardiovascular events: a literature update.红斑狼疮患者的现代治疗必须包括对其动脉粥样硬化性心血管事件高发生率的适当管理：文献综述。
Lupus Sci Med. 2025 Apr 8;12(1):e001160. doi: 10.1136/lupus-2024-001160.
7
The Atherosclerotic Plaque Microenvironment as a Therapeutic Target.作为治疗靶点的动脉粥样硬化斑块微环境
Curr Atheroscler Rep. 2025 Apr 2;27(1):47. doi: 10.1007/s11883-025-01294-y.
8
The cardioprotective role of the G protein-coupled receptor FFAR4 in atherosclerosis is independent of macrophage foam cell regulation.G蛋白偶联受体FFAR4在动脉粥样硬化中的心脏保护作用独立于巨噬细胞泡沫细胞调节。
J Biol Chem. 2025 May;301(5):108463. doi: 10.1016/j.jbc.2025.108463. Epub 2025 Mar 27.
9
Sphingolipid metabolites involved in the pathogenesis of atherosclerosis: perspectives on sphingolipids in atherosclerosis.参与动脉粥样硬化发病机制的鞘脂代谢产物：动脉粥样硬化中鞘脂的研究视角
Cell Mol Biol Lett. 2025 Feb 7;30(1):18. doi: 10.1186/s11658-024-00679-2.
10
Inflammation in atherosclerosis: a Big Idea that has underperformed so far.动脉粥样硬化中的炎症：一个至今表现欠佳的重大理念。
Curr Opin Lipidol. 2025 Apr 1;36(2):78-87. doi: 10.1097/MOL.0000000000000973. Epub 2025 Jan 22.
本文引用的文献
1
The human aorta; sulfate-containing polyuronides and the deposition of cholesterol.人类主动脉；含硫酸盐的聚糖醛酸苷与胆固醇的沉积
Arch Pathol (Chic). 1949 Oct;48(4):342-50.
2
Sites of vascular vulnerability in dogs demonstrated by Evans blue.
AMA Arch Pathol. 1957 Sep;64(3):303-11.
3
Lipoprotein lipase correlates positively and hepatic lipase inversely with calcific atherosclerosis in homozygous familial hypercholesterolemia.在纯合子家族性高胆固醇血症中，脂蛋白脂肪酶与钙化性动脉粥样硬化呈正相关，而肝脂肪酶与之呈负相关。
Arterioscler Thromb Vasc Biol. 1997 Feb;17(2):354-64. doi: 10.1161/01.atv.17.2.354.
4
Platelet-derived growth factor B chain promoter contains a cis-acting fluid shear-stress-responsive element.血小板衍生生长因子B链启动子含有一个顺式作用的流体切应力反应元件。
Proc Natl Acad Sci U S A. 1993 May 15;90(10):4591-5. doi: 10.1073/pnas.90.10.4591.
5
Macrophage and foam cell release of matrix-bound growth factors. Role of plasminogen activation.巨噬细胞和泡沫细胞释放基质结合生长因子。纤溶酶原激活的作用。
J Biol Chem. 1993 Jun 5;268(16):11951-8.
6
Proliferation of human smooth muscle cells promoted by lipoprotein(a).脂蛋白(a)促进人平滑肌细胞增殖。
Science. 1993 Jun 11;260(5114):1655-8. doi: 10.1126/science.8503012.
7
Effects of lipoprotein(a) on in vitro lysis of whole blood thrombi from healthy volunteers.
Thromb Res. 1993 Mar 1;69(5):479-87. doi: 10.1016/0049-3848(93)90236-h.
8
Regulation of adenine nucleotide concentration at endothelium-fluid interface by viscous shear flow.粘性剪切流对内皮-流体界面腺嘌呤核苷酸浓度的调节
Biophys J. 1993 Apr;64(4):1323-30. doi: 10.1016/S0006-3495(93)81498-X.
9
Nifedipine inhibits accumulation of LDL and cholesterol in the aorta of the normocholesterolemic rabbit.
Arterioscler Thromb. 1993 May;13(5):637-9. doi: 10.1161/01.atv.13.5.637.
10
The pathogenesis of atherosclerosis: a perspective for the 1990s.动脉粥样硬化的发病机制：20世纪90年代的展望
Nature. 1993 Apr 29;362(6423):801-9. doi: 10.1038/362801a0.
Zotero 插件