Oxidised low density lipoproteins and atherogenesis.
作者信息
Leake D S
出版信息
Br Heart J. 1993 Jun;69(6):476-8. doi: 10.1136/hrt.69.6.476.
Abstract
摘要
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Oxidised low density lipoproteins and atherogenesis.氧化型低密度脂蛋白与动脉粥样硬化的发生
Br Heart J. 1993 Jun;69(6):476-8. doi: 10.1136/hrt.69.6.476.
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Biological modification of lipoproteins and its role in atherogenesis.脂蛋白的生物修饰及其在动脉粥样硬化发生中的作用。
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Chlamydomonas reinhardtii chloroplasts express an orally immunogenic protein targeting the p210 epitope implicated in atherosclerosis immunotherapies.莱茵衣藻叶绿体表达一种口服免疫原性蛋白，靶向与动脉粥样硬化免疫治疗相关的 p210 表位。
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本文引用的文献
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Bovine aortic endothelial cells display macrophage-like properties towards acetylated 125I-labelled low density lipoprotein.牛主动脉内皮细胞对乙酰化的125I标记的低密度脂蛋白表现出巨噬细胞样特性。
Biochim Biophys Acta. 1980 Dec 5;620(3):631-5. doi: 10.1016/0005-2760(80)90155-1.
2
Enhanced macrophage degradation of biologically modified low density lipoprotein.生物修饰低密度脂蛋白的巨噬细胞降解增强。
Arteriosclerosis. 1983 Mar-Apr;3(2):149-59. doi: 10.1161/01.atv.3.2.149.
3
Modification of low density lipoprotein by endothelial cells involves lipid peroxidation and degradation of low density lipoprotein phospholipids.内皮细胞对低密度脂蛋白的修饰涉及脂质过氧化和低密度脂蛋白磷脂的降解。
Proc Natl Acad Sci U S A. 1984 Jun;81(12):3883-7. doi: 10.1073/pnas.81.12.3883.
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Monocytic origin of foam cells in human atherosclerotic plaques.人类动脉粥样硬化斑块中泡沫细胞的单核细胞起源。
Atherosclerosis. 1984 Dec;53(3):265-71. doi: 10.1016/0021-9150(84)90127-8.
5
Enhanced macrophage degradation of low density lipoprotein previously incubated with cultured endothelial cells: recognition by receptors for acetylated low density lipoproteins.经培养的内皮细胞预先孵育的低密度脂蛋白，其巨噬细胞降解作用增强：通过乙酰化低密度脂蛋白受体识别。
Proc Natl Acad Sci U S A. 1981 Oct;78(10):6499-503. doi: 10.1073/pnas.78.10.6499.
6
Macrophage oxidation of low density lipoprotein generates a modified form recognized by the scavenger receptor.巨噬细胞对低密度脂蛋白的氧化作用会产生一种可被清道夫受体识别的修饰形式。
Arteriosclerosis. 1986 Sep-Oct;6(5):505-10. doi: 10.1161/01.atv.6.5.505.
7
Antiatherogenic effect of probucol unrelated to its hypocholesterolemic effect: evidence that antioxidants in vivo can selectively inhibit low density lipoprotein degradation in macrophage-rich fatty streaks and slow the progression of atherosclerosis in the Watanabe heritable hyperlipidemic rabbit.普罗布考的抗动脉粥样硬化作用与其降胆固醇作用无关：体内抗氧化剂可选择性抑制富含巨噬细胞的脂肪条纹中低密度脂蛋白降解并减缓渡边遗传性高脂血症兔动脉粥样硬化进展的证据。
Proc Natl Acad Sci U S A. 1987 Nov;84(21):7725-9. doi: 10.1073/pnas.84.21.7725.
8
Probucol prevents the progression of atherosclerosis in Watanabe heritable hyperlipidemic rabbit, an animal model for familial hypercholesterolemia.普罗布考可阻止渡边遗传性高脂血症兔（一种家族性高胆固醇血症动物模型）动脉粥样硬化的进展。
Proc Natl Acad Sci U S A. 1987 Aug;84(16):5928-31. doi: 10.1073/pnas.84.16.5928.
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Evidence for the presence of oxidatively modified low density lipoprotein in atherosclerotic lesions of rabbit and man.兔和人动脉粥样硬化病变中存在氧化修饰低密度脂蛋白的证据。
J Clin Invest. 1989 Oct;84(4):1086-95. doi: 10.1172/JCI114271.
10
Lack of effect of probucol on atheroma formation in cholesterol-fed rabbits kept at comparable plasma cholesterol levels.在血浆胆固醇水平相当的情况下，普罗布考对喂食胆固醇的兔子动脉粥样硬化斑块形成没有影响。
Atherosclerosis. 1989 Feb;75(2-3):145-55. doi: 10.1016/0021-9150(89)90171-8.
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