David C A, Peerless S J
Department of Neurological Surgery, University of Miami/Jackson Memorial Medical Center, Florida, USA.
Neurosurgery. 1995 Mar;36(3):588-90. doi: 10.1227/00006123-199503000-00020.
We report a case of a cerebral arteriovenous malformation presenting as a pseudotumor cerebri syndrome. The lack of mass effect, hemorrhage, or hydrocephalus, in addition to angiographic evidence of high-flow and venous outflow restriction, implicate increased cerebral blood flow and venous hypertension as the mechanism for the papilledema. This case illustrates a pathophysiological mechanism in the development of intracranial hypertension associated with vascular malformations.
我们报告一例表现为假性脑瘤综合征的脑动静脉畸形病例。除了高流量和静脉流出受限的血管造影证据外,缺乏占位效应、出血或脑积水,提示脑血流量增加和静脉高压是视乳头水肿的机制。该病例说明了与血管畸形相关的颅内高压发展中的一种病理生理机制。