Major histocompatibility complex class I antigens and the control of viral infections by natural killer cells.
作者信息
Brutkiewicz R R, Welsh R M
机构信息
Department of Pathology, University of Massachusetts Medical Center, Worcester 01655-0125, USA.
出版信息
J Virol. 1995 Jul;69(7):3967-71. doi: 10.1128/JVI.69.7.3967-3971.1995.
Abstract
摘要
相似文献
1
Major histocompatibility complex class I antigens and the control of viral infections by natural killer cells.主要组织相容性复合体I类抗原与自然杀伤细胞对病毒感染的控制
J Virol. 1995 Jul;69(7):3967-71. doi: 10.1128/JVI.69.7.3967-3971.1995.
2
The first molecular basis of the "missing self" hypothesis.
J Immunol. 2006 Nov 1;177(9):5759-60. doi: 10.4049/jimmunol.177.9.5759.
3
[NK cells and NKT cells].[自然杀伤细胞和自然杀伤T细胞]
Nihon Rinsho. 2005 Apr;63 Suppl 4:159-65.
4
Induction, control and consequences of virus specific cytotoxic T cells.病毒特异性细胞毒性T细胞的诱导、控制及后果。
Immunol Rev. 1981;58:157-80. doi: 10.1111/j.1600-065x.1981.tb00353.x.
5
What is a natural killer cell?
Nat Immunol. 2002 Jan;3(1):6-8. doi: 10.1038/ni0102-6.
6
Major histocompatibility complex class I-independent killing of xenogeneic targets by rat allospecific natural killer cells.
Transplantation. 1997 Jan 15;63(1):119-23. doi: 10.1097/00007890-199701150-00022.
7
Recent advances in viral evasion of the MHC Class I processing pathway.病毒逃避 MHC I 加工途径的最新进展。
Curr Opin Immunol. 2016 Jun;40:43-50. doi: 10.1016/j.coi.2016.02.007. Epub 2016 Apr 8.
8
Ly-49-independent inhibition of natural killer cell-mediated cytotoxicity by a soluble major histocompatibility complex class I molecule.可溶性主要组织相容性复合体I类分子对自然杀伤细胞介导的细胞毒性的Ly-49非依赖性抑制作用。
Eur J Immunol. 1994 Sep;24(9):2110-4. doi: 10.1002/eji.1830240927.
9
Selection and adaptation of cells expressing major histocompatibility complex class I-specific receptors of the natural killer complex.自然杀伤复合体中主要组织相容性复合体I类特异性受体表达细胞的选择与适应性
J Exp Med. 1997 Aug 4;186(3):349-51. doi: 10.1084/jem.186.3.349.
10
[Natural cytotoxic lymphocytes (natural killers) and their role in resistance to viral infections].[自然细胞毒性淋巴细胞（自然杀伤细胞）及其在抗病毒感染中的作用]
Vopr Virusol. 1987 Mar-Apr;32(2):142-51.
引用本文的文献
1
MHC class I expression in intestinal cells is reduced by rotavirus infection and increased in bystander cells lacking rotavirus antigen.轮状病毒感染会降低肠道细胞中 MHC Ⅰ类分子的表达，而在缺乏轮状病毒抗原的旁观者细胞中则会增加。
Sci Rep. 2018 Jan 8;8(1):67. doi: 10.1038/s41598-017-18464-x.
2
Preclinical Mouse Models for Analysis of the Therapeutic Potential of Engineered Oncolytic Herpes Viruses.用于分析工程化溶瘤疱疹病毒治疗潜力的临床前小鼠模型
ILAR J. 2016;57(1):63-72. doi: 10.1093/ilar/ilw002.
3
Specific dysregulation of IFNγ production by natural killer cells confers susceptibility to viral infection.自然杀伤细胞对γ干扰素产生的特异性失调会使人易患病毒感染。
PLoS Pathog. 2014 Dec 4;10(12):e1004511. doi: 10.1371/journal.ppat.1004511. eCollection 2014 Dec.
4
NK cells and virus-related cancers.自然杀伤细胞与病毒相关癌症。
Crit Rev Oncog. 2014;19(1-2):107-19. doi: 10.1615/critrevoncog.2014010866.
5
Sorting Out Pandora's Box: Discerning the Dynamic Roles of Liver Microenvironment in Oncolytic Virus Therapy for Hepatocellular Carcinoma.理清潘多拉魔盒：洞悉肝脏微环境在肝细胞癌溶瘤病毒治疗中的动态作用
Front Oncol. 2014 Apr 22;4:85. doi: 10.3389/fonc.2014.00085. eCollection 2014.
6
Evolving role of 2B4/CD244 in T and NK cell responses during virus infection.2B4/CD244 在病毒感染过程中 T 和 NK 细胞反应中的作用不断演变。
Front Immunol. 2012 Dec 11;3:377. doi: 10.3389/fimmu.2012.00377. eCollection 2012.
7
NK cells controlling virus-specific T cells: Rheostats for acute vs. persistent infections.NK 细胞控制病毒特异性 T 细胞：急性与持续性感染的调节开关。
Virology. 2013 Jan 5;435(1):37-45. doi: 10.1016/j.virol.2012.10.005.
8
Role of human cytomegalovirus genotype polymorphisms in AIDS patients with cytomegalovirus retinitis.人巨细胞病毒基因型多态性在 AIDS 合并巨细胞病毒性视网膜炎患者中的作用。
Med Microbiol Immunol. 2013 Feb;202(1):37-47. doi: 10.1007/s00430-012-0244-3. Epub 2012 Jun 6.
9
Modulation of human mesenchymal stem cell immunogenicity through forced expression of human cytomegalovirus us proteins.通过强制表达人巨细胞病毒 us 蛋白调节人间充质干细胞的免疫原性。
PLoS One. 2012;7(5):e36163. doi: 10.1371/journal.pone.0036163. Epub 2012 May 30.
10
Genetic polymorphism analysis of killer cell immunoglobulin-like receptor genes in the Chinese Uygur population.中国维吾尔族人群杀伤细胞免疫球蛋白样受体基因遗传多态性分析。
Mol Biol Rep. 2012 Mar;39(3):3017-28. doi: 10.1007/s11033-011-1064-z. Epub 2011 Jun 24.
本文引用的文献
1
A case for cytokines as effector molecules in the resolution of virus infection.细胞因子作为病毒感染消退中的效应分子的实例。
Immunol Today. 1993 Apr;14(4):155-7. doi: 10.1016/0167-5699(93)90277-R.
2
3
Human cytomegalovirus down-regulates HLA class I expression by reducing the stability of class I H chains.人类巨细胞病毒通过降低I类重链的稳定性来下调HLA I类分子的表达。
J Immunol. 1993 Nov 1;151(9):4455-64.
4
Modulation of major histocompatibility complex antigen expression by viral infection.病毒感染对主要组织相容性复合体抗原表达的调节作用。
Am J Pathol. 1994 Apr;144(4):637-50.
5
HLA class I molecules are not transported to the cell surface in cells infected with herpes simplex virus types 1 and 2.
J Immunol. 1994 Mar 15;152(6):2736-41.
6
Ly-49 mediates EL4 lymphoma adhesion to isolated class I major histocompatibility complex molecules.Ly-49介导EL4淋巴瘤与分离出的I类主要组织相容性复合体分子的黏附。
J Exp Med. 1994 Mar 1;179(3):1011-5. doi: 10.1084/jem.179.3.1011.
7
Down-regulation of major histocompatibility complex class I synthesis by murine cytomegalovirus early gene expression.小鼠巨细胞病毒早期基因表达对主要组织相容性复合体I类合成的下调作用。
J Virol. 1994 Mar;68(3):1805-11. doi: 10.1128/JVI.68.3.1805-1811.1994.
8
A natural killer cell receptor specific for a major histocompatibility complex class I molecule.一种对主要组织相容性复合体I类分子具有特异性的自然杀伤细胞受体。
J Exp Med. 1994 Aug 1;180(2):687-92. doi: 10.1084/jem.180.2.687.
9
Human natural killer cell receptors for HLA-class I molecules. Evidence that the Kp43 (CD94) molecule functions as receptor for HLA-B alleles.人类HLA - I类分子的自然杀伤细胞受体。Kp43（CD94）分子作为HLA - B等位基因受体的证据。
J Exp Med. 1994 Aug 1;180(2):545-55. doi: 10.1084/jem.180.2.545.
10
NKB1: a natural killer cell receptor involved in the recognition of polymorphic HLA-B molecules.NKB1：一种参与识别多态性HLA - B分子的自然杀伤细胞受体。
J Exp Med. 1994 Aug 1;180(2):537-43. doi: 10.1084/jem.180.2.537.
Zotero 插件