Abstract
This review stresses particularly the role that "intercellular" antigens and antibodies, complement and lymphocytic dysfunction may play in pemphigus. In addition evidences in the literature in favour of exogenic factors inducing pemphigus are surveyed. Further an attempt is made to summarize the possibilities for the etiology and the blister formation in this disease.
摘要
本综述特别强调了“细胞间”抗原和抗体、补体及淋巴细胞功能障碍在天疱疮中可能发挥的作用。此外，还综述了文献中支持外源性因素诱发天疱疮的证据。进一步尝试总结该疾病病因及水疱形成的可能性。
相似文献
1
[Pemphigus].[天疱疮]
Hautarzt. 1976 Jun;27(6):253-60.
2
Pemphigus. Facts, unanswered questions, and speculations.天疱疮。事实、未解决的问题及推测。
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3
Pemphigus vegetans of Hallopeau: immunofluorescent studies.哈洛佩奥增殖性天疱疮：免疫荧光研究
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4
[Complement-independent bulla formation in pemphigus vulgaris by IgG4].
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5
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7
Exacerbation of pemphigus after influenza vaccination.流感疫苗接种后天疱疮病情加重。
Clin Exp Dermatol. 2008 Nov;33(6):718-20. doi: 10.1111/j.1365-2230.2008.02835.x. Epub 2008 Aug 2.
8
Pemphigus.
Br J Hosp Med. 1987 Apr;37(4):326-7, 330-1, 334.
9
Further support for a role for Th2-like cytokines in blister formation of pemphigus.对Th2样细胞因子在天疱疮水疱形成中作用的进一步支持。
Clin Immunol. 2001 Feb;98(2):264-71. doi: 10.1006/clim.2000.4974.
10
[Cellular immunity in the pathogenesis of pemphigus vulgaris].[寻常型天疱疮发病机制中的细胞免疫]
Z Haut Geschlechtskr. 1972 Feb 1;47(3):95-8.
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