Rice L E, Levin M L, Jennings R B, Ivanovich P
Nephron. 1976;17(4):279-87. doi: 10.1159/000180732.
Renovascular hypertension developed in an anephric 37-year-old patient after he received a cadaveric renal transplant from a 2-year-old donor. Despite adequate homograft function, a transplant nephrectomy was perfomed because of intractable, lifethreatening hypertension. There was relative stenosis throughout the course of the transplanted renal artery. Pathologic examination of the kidney did not demonstrate evidence of technical failure or immunological or hypertensive damage. Atrophic changes in the media of the renal artery may have resulted from radiation damage. The hypertension appears to have been caused by disproportionate growth between the parenchyma in the hypertrophying pediatric homograft and its renal artery.
一名37岁的无肾患者在接受了一名2岁供体的尸体肾移植后发生了肾血管性高血压。尽管同种异体移植肾功能良好，但由于顽固性、危及生命的高血压，还是进行了移植肾切除术。移植肾动脉全程存在相对狭窄。对肾脏的病理检查未发现技术失误、免疫或高血压损伤的证据。肾动脉中层的萎缩性改变可能是辐射损伤所致。高血压似乎是由于肥大的小儿同种异体移植肾实质与其肾动脉之间生长不均衡所致。
