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[眼内注入硅油后继发性青光眼的发病机制]

[Pathogenesis of secondary glaucoma after intraocular silicone oil administration].

作者信息

Slezak H, Haddad R, Scholda C

机构信息

2. Augenklinik der Universität Wien.

出版信息

Klin Monbl Augenheilkd. 1994 Nov;205(5):298-300. doi: 10.1055/s-2008-1045532.

DOI:10.1055/s-2008-1045532
PMID:7844945
Abstract

BACKGROUND

There are various causes of increasing ocular tension after vitrectomy and intraocular silicone oil filling; it is reported on a new type of postoperative secondary glaucoma.

MATERIALS AND METHODS

A 56-year-old man with flat anterior chamber and secondary glaucoma after vitrectomy and intraocular silicone oil filling was examined; only the transpupillary inspection of the posterior surface of the iris with the three mirror contact glass yielded the decisive reference to the pathogenesis of the glaucoma.

RESULTS

The secondary glaucoma was caused by small perfluoroctane bubbles blocking up Ando's peripheral iris coloboma.

CONCLUSIONS

In case of an unclear pathogenesis of secondary glaucoma after vitrectomy and fluid/perfluoroctane/silicone oil exchange among others a blockade of Ando's peripheral coloboma by small perfluoroctane bubbles should be considered as a cause of increased intraocular tension.

摘要

背景

玻璃体切除联合眼内硅油填充术后眼压升高有多种原因;有关于一种新型术后继发性青光眼的报道。

材料与方法

对一名56岁男性进行检查,该患者在玻璃体切除联合眼内硅油填充术后出现前房扁平及继发性青光眼;仅通过三面镜接触镜对虹膜后表面进行经瞳孔检查,就为青光眼的发病机制提供了决定性依据。

结果

继发性青光眼是由小的全氟辛烷气泡阻塞安藤周边虹膜缺损所致。

结论

在玻璃体切除术后及进行液体/全氟辛烷/硅油置换等情况下,如果继发性青光眼的发病机制不明,应考虑小的全氟辛烷气泡阻塞安藤周边缺损是眼压升高的一个原因。

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1
[Pathogenesis of secondary glaucoma after intraocular silicone oil administration].[眼内注入硅油后继发性青光眼的发病机制]
Klin Monbl Augenheilkd. 1994 Nov;205(5):298-300. doi: 10.1055/s-2008-1045532.
2
[Removal of silicone oil in treatment of so-called emulsification glaucoma].[硅油取出术治疗所谓的乳化性青光眼]
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