Lubowski D Z, King D W
Colorectal Unit, St George Hospital, Sydney, New South Wales, Australia.
Aust N Z J Surg. 1995 Feb;65(2):87-92. doi: 10.1111/j.1445-2197.1995.tb07267.x.
Obstructed defecation poses a challenging clinical problem and in many patients presenting with this syndrome the underlying pathophysiology cannot be determined. Up to now, attempts to diagnose and treat obstructed defecation (anismus) have focused on the function of the somatic pelvic floor musculature surrounding the anorectum, and concepts such as 'puborectalis paradox' and 'spastic pelvic floor' have gained widespread acceptance despite there being no objective data to support such concepts. New evidence showing that defecation is an integrated process of colonic and rectal emptying suggests that anismus may be much more complex than a simple disorder of the pelvic floor muscles. In a small number of patients obstructed defecation is caused by a more simple mechanism, such as internal sphincter hypertonia or a large rectocele, which is easily corrected surgically. Careful selection of patients for treatment, based on identifying the underlying pathophysiological disorder, is emphasized.
排便障碍是一个具有挑战性的临床问题,许多患有该综合征的患者其潜在的病理生理学机制尚无法确定。到目前为止,诊断和治疗排便障碍(盆底失弛缓综合征)的尝试主要集中在围绕肛门直肠的躯体盆底肌肉组织的功能上,尽管没有客观数据支持,但诸如“耻骨直肠肌矛盾”和“盆底痉挛”等概念已被广泛接受。新的证据表明,排便是结肠和直肠排空的一个综合过程,这表明盆底失弛缓综合征可能比单纯的盆底肌肉紊乱要复杂得多。在少数患者中,排便障碍是由更简单的机制引起的,如内括约肌张力过高或巨大直肠膨出,这些情况通过手术很容易纠正。强调要基于识别潜在的病理生理紊乱来仔细挑选治疗患者。