Danilova N N, Korshunova S G, Sokolov E N
Zh Vyssh Nerv Deiat Im I P Pavlova. 1994 Nov-Dec;44(6):932-43.
Heart rate and respiration were recorded in a group of 90 subjects (25 males and 65 females) aged 17-19 during rest and under informational load (arithmetical tasks) lasting 3 min each. Off-line spectral analysis was performed for all the subjects. Anxiety according to Spilberger and strength of excitation-inhibition according to Strelau were also tested. It was shown that heart rate increased significantly in the group as a whole, however, variability of RR-intervals remained unchanged. Then two subgroups of subjects who responded to information load by a decrease and increase of RR-interval variability were distinguished. These subgroups were characterized respectively by the high and low levels of personal anxiety. The decrease of RR-interval variability in the high-anxiety subgroup was associated with a decrease of power in all frequency bands of the rate spectrum. The increase of RR-interval variability in the low-anxiety subground was due to an increase of heart rate modulation in a low-frequency band of the heart rate spectrum. Fatigue is regarded as a cause of such heart rate modulation.
对90名年龄在17至19岁之间的受试者（25名男性和65名女性）在休息状态以及在持续3分钟的信息负荷（算术任务）状态下的心率和呼吸进行了记录。对所有受试者进行了离线频谱分析。还测试了根据斯皮尔伯格量表得出的焦虑程度以及根据斯特劳量表得出的兴奋抑制强度。结果显示，整个组的心率显著增加，然而，RR间期的变异性保持不变。然后区分出了两个亚组，一组受试者的RR间期变异性对信息负荷的反应是降低，另一组是升高。这些亚组分别以个人焦虑的高水平和低水平为特征。高焦虑亚组中RR间期变异性的降低与心率频谱所有频段功率的降低有关。低焦虑亚组中RR间期变异性的增加是由于心率频谱低频段中心率调制的增加。疲劳被认为是这种心率调制的一个原因。
