Smetnev A S, Dobrotvorskaia T E
Kardiologiia. 1976 Jul;16(7):5-11.
The presented analysis deals with the physiopathological mechanisms of the debelopment of postinfarction cardiac insufficiency and the clinical peculiarities of its manifestations. It is emphasized that the leading cause of cardiac insufficiency consists in a reduction of the contractile function of the left ventricular myocardium due to the development of asynergy in the cicatrical zone. The addition of several accompanying factors, such as heart mitralization, tachysystolic form of ventricular fibrillation, repeated infarction with a growing asynergic zone, aggravate the course of cardiac insufficiency providing for the development of hypertension in the general circulation system.
所呈现的分析涉及心肌梗死后心脏功能不全发展的生理病理机制及其临床表现的临床特点。强调心脏功能不全的主要原因在于瘢痕区协同失调的发展导致左心室心肌收缩功能降低。多种伴随因素的叠加，如心脏二尖瓣化、室性心动过速形式的心室颤动、梗死灶扩大且协同失调区域增大的反复梗死，会加重心脏功能不全的病程，进而导致体循环系统高血压的发生。
