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Glycogen degradation by adrenergic agonists and glucagon in periportal and perivenous rat hepatocyte cultures.

作者信息

Tosh D, Agius L

机构信息

Department of Medicine, Medical School, University of Newcastle-upon-Tyne, UK.

出版信息

Biochim Biophys Acta. 1994 Apr 28;1221(3):238-42. doi: 10.1016/0167-4889(94)90246-1.

DOI:10.1016/0167-4889(94)90246-1
PMID:7909448
Abstract

The stimulation of glycogen degradation by adrenergic agonists and glucagon was determined in hepatocytes isolated from the periportal and perivenous zones of rat liver and maintained in culture. Glucagon, epinephrine and phenylephrine (an alpha-adrenergic receptor agonist) caused a greater stimulation of glycogen degradation in periportal than in perivenous hepatocytes, whereas isoproterenol (a beta-adrenergic receptor agonist) caused a similar degree of glycogenolysis in the two cell populations. The results suggest that the hormonal stimulation of glycogen degradation differs in periportal and perivenous hepatocytes.

摘要

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