Andres J
Katedry Anestezjologii i Intensywnej Terapii Collegium Medicum Uniwersytetu Jagiellońskiego, Kraków.
Przegl Lek. 1994;51(5):213-6.
Reversible contractile dysfunction ("myocardial stunning") now is recognized in laboratory animals as well as in the clinic. The precise mechanism(s) responsible for this transient decrease in myocardial contractility is at present unknown. However substantial body of experimental evidence suggest failure in calcium homeostasis due to free radicals injury and calcium overload in ischemic/reperfused myocardium.
可逆性收缩功能障碍（“心肌顿抑”）目前在实验动物和临床中均已得到确认。导致心肌收缩力这种短暂下降的确切机制目前尚不清楚。然而，大量实验证据表明，缺血/再灌注心肌中自由基损伤和钙超载导致钙稳态失衡。
