Ichikawa H, Fukui T, Negishi A, Sugita K, Kanda M
Department of Neurology, Showa University School of Medicine.
Rinsho Shinkeigaku. 1994 Jun;34(6):569-76.
We reported a patient with bilateral cerebellar peduncle infarcts who had an abrupt onset of bilateral hearing loss. A hypertensive 56-year-old man suddenly experienced bilateral hearing loss without other accompanying neurological deficits. He was hospitalized and treated for "idiopathic deafness". In addition, dysarthria and ataxic gait appeared two days later and he was transferred to our hospital. On neurological examination, the patient presented with diplopia, neurosensory hearing loss (approximately 70 dB) ataxic dysarthria, bilateral cerebellar ataxia and bilateral Babinski's signs. Auditory brain stem evoked response demonstrated prolonged delay of interpeak latency between waves III-IV. CT and MRI revealed fresh ischemic lesions symmetrically located at the middle cerebellar peduncles and cerebellar medullary body. Cerebral angiography showed total occlusion of the left vertebral artery and a stenotic right vertebral artery at the ostium of the posterior inferior cerebellar artery. We postulated that hearing impairment in this patient resulted from transient ischemia of the bilateral auditory tract in the brain stem or the peripheral cochlear system, but the definitive cause of the transient hearing loss remains undetermined. Concomitant appearance of a symmetrical infarction at the cerebellar peduncles is rare. We suggest that a circulation defect involving a multivascular system, which resulted in "border zone infarction" occurred at these regions.
我们报告了一例双侧小脑脚梗死患者,该患者突然出现双侧听力丧失。一名56岁的高血压男性突然出现双侧听力丧失,无其他伴随的神经功能缺损。他因“特发性耳聋”住院治疗。此外,两天后出现构音障碍和共济失调步态,随后被转至我院。神经系统检查时,患者表现为复视、感音神经性听力丧失(约70分贝)、共济失调性构音障碍、双侧小脑共济失调和双侧巴宾斯基征。听性脑干诱发电位显示III-IV波间峰潜伏期延长。CT和MRI显示新鲜缺血性病变对称位于小脑中间脚和小脑髓体。脑血管造影显示左椎动脉完全闭塞,右椎动脉在小脑后下动脉开口处狭窄。我们推测该患者的听力障碍是由于脑干双侧听觉通路或外周耳蜗系统的短暂缺血所致,但短暂性听力丧失的确切原因仍未确定。小脑脚同时出现对称性梗死很少见。我们认为,涉及多血管系统的循环缺陷导致了这些区域出现“边缘带梗死”。