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The E6 protein of human papillomavirus type 16 functions as a transcriptional repressor in a mechanism independent of the tumor suppressor protein, p53.

作者信息

Etscheid B G, Foster S A, Galloway D A

机构信息

Fred Hutchinson Cancer Research Center, Seattle, Washington 98104.

出版信息

Virology. 1994 Dec;205(2):583-5. doi: 10.1006/viro.1994.1684.

DOI:10.1006/viro.1994.1684
PMID:7975261
Abstract

The E6 protein of human papillomavirus (HPV) type 16 displays a number of activities when transfected into cultured cells, including transcriptional activation of several viral promoters and targeting of p53 for degradation. HPV 16E6 was found to function as a transcriptional repressor of the moloney murine leukemia virus long terminal repeat and the cytomegalovirus immediate early promoter. Although the degree of transcriptional repression was low, a dose-dependent two- to threefold decrease in promoter activity was consistently seen in cells expressing 16E6. HPV 16E6-dependent transcriptional repression was observed in C33a cells, which express mutant p53, and in Saos-2 cells, which lack p53. These results indicate that 16E6-dependent repression of promoter activity is unlikely to be mediated by p53.

摘要

相似文献

1
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2
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The human papillomavirus (HPV) E6* proteins from high-risk, mucosal HPVs can direct degradation of cellular proteins in the absence of full-length E6 protein.来自高危黏膜型人乳头瘤病毒(HPV)的E6*蛋白在缺乏全长E6蛋白的情况下可直接导致细胞蛋白降解。
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p53 status dictates responses of B lymphomas to monotherapy with proteasome inhibitors.
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J Virol. 2001 May;75(9):4467-72. doi: 10.1128/JVI.75.9.4467-4472.2001.
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