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Ischemia potentiates the mechanosensitive modulation of atrial ATP-sensitive potassium channels.

作者信息

Van Wagoner D R, Lamorgese M

机构信息

Cardiovascular Biology, Research Institute, FFI, Cleveland Clinic Foundation, Ohio 44195-5069.

出版信息

Ann N Y Acad Sci. 1994 Jun 17;723:392-5.

PMID:8030893
Abstract
摘要

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Ischemia potentiates the mechanosensitive modulation of atrial ATP-sensitive potassium channels.缺血增强心房ATP敏感性钾通道的机械敏感性调节。
Ann N Y Acad Sci. 1994 Jun 17;723:392-5.
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Mechanosensitive gating of atrial ATP-sensitive potassium channels.心房ATP敏感性钾通道的机械敏感性门控
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Pinacidil but not nicorandil opens ATP-sensitive K+ channels and protects against simulated ischemia in rabbit myocytes.匹那地尔而非尼可地尔可开放ATP敏感性钾通道，并对兔心肌细胞的模拟缺血具有保护作用。
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[Potential role of ATP-sensitive K+ channels in ischemia- and reperfusion-induced arrhythmias].[ATP敏感性钾通道在缺血及再灌注诱导的心律失常中的潜在作用]
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Mitochondrial K(ATP) channel opening is important during index ischemia and following myocardial reperfusion in ischemic preconditioned rat hearts.线粒体ATP敏感性钾通道开放在缺血预处理大鼠心脏的初次缺血期间及心肌再灌注后具有重要作用。
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An explanation for the reported observation that ATP dependent potassium channel openers fail to mimic preconditioning.
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