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Calcium and inositol 1,4,5-trisphosphate-induced Ca2+ release.

作者信息

Combettes L, Champeil P

出版信息

Science. 1994 Aug 5;265(5173):813-5. doi: 10.1126/science.8047889.

DOI:10.1126/science.8047889
PMID:8047889
Abstract
摘要

相似文献

1
Calcium and inositol 1,4,5-trisphosphate-induced Ca2+ release.钙与肌醇1,4,5-三磷酸诱导的钙离子释放。
Science. 1994 Aug 5;265(5173):813-5. doi: 10.1126/science.8047889.
2
The role of Ca2+ feedback in shaping InsP3-evoked Ca2+ signals in mouse pancreatic acinar cells.Ca2+ 反馈在塑造小鼠胰腺腺泡细胞中 InsP3 诱发的 Ca2+ 信号方面的作用。
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3
Small-conductance Ca2+-dependent K+ channels are the target of spike-induced Ca2+ release in a feedback regulation of pyramidal cell excitability.小电导钙依赖性钾通道是锥体细胞兴奋性反馈调节中动作电位诱导的钙释放的靶点。
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4
Regulatory and spatial aspects of inositol trisphosphate-mediated calcium signals.肌醇三磷酸介导的钙信号的调节及空间方面
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Quantal responses to inositol 1,4,5-trisphosphate are not a consequence of Ca2+ regulation of inositol 1,4,5-trisphosphate receptors.对肌醇1,4,5-三磷酸的量子反应并非由钙离子对肌醇1,4,5-三磷酸受体的调节所致。
Biochem J. 1995 Dec 15;312 ( Pt 3)(Pt 3):789-94. doi: 10.1042/bj3120789.
6
Dynamic control of inositol 1,4,5-trisphosphate-induced Ca2+ release: a theoretical explanation for the quantal release of Ca2+.
Mol Pharmacol. 1992 Jan;41(1):110-4.
7
[Ca2+]i-sensitive, IP3-independent Ca2+ influx in smooth muscle of rat vas deferens revealed by procaine.普鲁卡因揭示大鼠输精管平滑肌中对细胞内钙离子浓度敏感、不依赖三磷酸肌醇的钙离子内流
Br J Pharmacol. 1993 Dec;110(4):1353-8. doi: 10.1111/j.1476-5381.1993.tb13968.x.
8
Quantal calcium release by purified reconstituted inositol 1,4,5-trisphosphate receptors.纯化重组的肌醇1,4,5-三磷酸受体引发的量子钙释放
Nature. 1992 Mar 26;356(6367):350-2. doi: 10.1038/356350a0.
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Magnesium sulfate inhibits the oxytocin-induced production of inositol 1,4,5-trisphosphate in cultured human myometrial cells.
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Control of IP(3)-mediated Ca2+ puffs in Xenopus laevis oocytes by the Ca2+-binding protein parvalbumin.通过钙结合蛋白小白蛋白对非洲爪蟾卵母细胞中IP(3)介导的钙瞬变的调控
J Physiol. 2001 Aug 15;535(Pt 1):3-16. doi: 10.1111/j.1469-7793.2001.t01-2-00003.x.

引用本文的文献

1
Inositol serves as a natural inhibitor of mitochondrial fission by directly targeting AMPK.肌醇通过直接靶向 AMPK 充当线粒体分裂的天然抑制剂。
Mol Cell. 2021 Sep 16;81(18):3803-3819.e7. doi: 10.1016/j.molcel.2021.08.025.
2
Inositol 1,4,5-trisphosphate [correction of tris-phosphate] activation of inositol trisphosphate [correction of tris-phosphate] receptor Ca2+ channel by ligand tuning of Ca2+ inhibition.通过对Ca2+抑制的配体调节实现1,4,5-三磷酸肌醇[纠正为三磷酸]对三磷酸肌醇[纠正为三磷酸]受体Ca2+通道的激活
Proc Natl Acad Sci U S A. 1998 Dec 22;95(26):15821-5. doi: 10.1073/pnas.95.26.15821.
3
Isoform-specific function of single inositol 1,4,5-trisphosphate receptor channels.
单肌醇1,4,5-三磷酸受体通道的亚型特异性功能
Biophys J. 1998 Aug;75(2):834-9. doi: 10.1016/S0006-3495(98)77572-1.
4
The effects of free [Ca2+] on the cytosolic face of the inositol (1,4,5)-trisphosphate receptor at the single channel level.游离[Ca2+]在单通道水平对肌醇（1,4,5）-三磷酸受体胞质面的影响。
Biochem J. 1998 Feb 15;330 ( Pt 1)(Pt 1):559-64. doi: 10.1042/bj3300559.
5
Ca2+ differentially regulates the ligand-affinity states of type 1 and type 3 inositol 1,4,5-trisphosphate receptors.钙离子对1型和3型肌醇1,4,5-三磷酸受体的配体亲和力状态具有差异性调节作用。
Biochem J. 1997 Mar 1;322 ( Pt 2)(Pt 2):591-6. doi: 10.1042/bj3220591.
6
Inositol 1,4,5-trisphosphate-induced Ca2+ release is regulated by cytosolic Ca2+ in intact skeletal muscle.在完整的骨骼肌中，肌醇1,4,5-三磷酸诱导的钙离子释放受胞质钙离子调控。
Pflugers Arch. 1996 Sep;432(5):782-90. doi: 10.1007/s004240050199.
7
Fast kinetics of calcium liberation induced in Xenopus oocytes by photoreleased inositol trisphosphate.光释放的肌醇三磷酸在非洲爪蟾卵母细胞中诱导的钙释放的快速动力学。
Biophys J. 1996 Jan;70(1):222-37. doi: 10.1016/S0006-3495(96)79565-6.
8
Local positive feedback by calcium in the propagation of intracellular calcium waves.钙在细胞内钙波传播中的局部正反馈。
Biophys J. 1995 Nov;69(5):1683-97. doi: 10.1016/S0006-3495(95)80086-X.