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野生兀鹫(矛隼)经皮暴露于对硫磷。

Percutaneous exposure to ethyl parathion in a feral Griffon vulture (Gyps fulvus).

作者信息

Shlosberg A, Lublin A, Bahat O, Heymann G D, Hanji V, Bellaiche M, Mechani S, Nyska A

机构信息

Kimron Veterinary Institute, Bet Dagan, Israel.

出版信息

Vet Hum Toxicol. 1994 Jun;36(3):224-6.

PMID:8066970
Abstract

A feral Griffon vulture (Gyps fulvus) was found with tremors, weakness, digit and wing flexion, and an inability to fly. A zero blood cholinesterase activity and a favorable response to treatment with pralidoxime hydrochloride indicated exposure to an anticholinergic pesticide. The bird died after 7 d, and traces of the organophosphate insecticide ethyl parathion were found in the liver and from a blue discolored skin area of the neck. Continuous exposure to ethyl parathion through dermal absorption was presumed the cause of death of the vulture.

摘要

一只野生的兀鹫(矛隼兀鹫)被发现出现震颤、虚弱、手指和翅膀弯曲以及无法飞行的症状。血液胆碱酯酶活性为零,且对盐酸氯解磷定治疗有良好反应,表明其接触了抗胆碱能农药。这只鸟在7天后死亡,在其肝脏以及颈部皮肤变蓝的区域发现了有机磷杀虫剂对硫磷的痕迹。推测通过皮肤吸收持续接触对硫磷是这只兀鹫死亡的原因。

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