Furusawa K, Yamaoka M, Ichikawa N
Oral and Maxillofacial Surgery Department II, Matsumoto Dental College Nagano, Japan.
Cleft Palate Craniofac J. 1994 May;31(3):161-6. doi: 10.1597/1545-1569_1994_031_0161_rosait_2.3.co_2.
The responsiveness of receptors supplying the oral mucosa to air pressures generated during consonant production was investigated to obtain information about hypothetical mechanisms underlying speech deficits. The delay between the onset of the neural discharge and the pop puff of phonation (mouth-exist pressure) for /pa/ production was significantly shorter and less variable than it was for /ta/ and /ka/ production, suggesting that the discharge is more closely coupled to the onset of /pa/ production. The data were interpreted to imply that single fibers of the infraorbital nerve respond to the build-up of oral air pressure during /pa/ production. This, and similar sensory information, may be used by the central neural mechanisms which monitor and control the air pressures required for phonation.
为了获取有关言语缺陷潜在假设机制的信息，研究了供应口腔黏膜的感受器对辅音产生过程中所产生气压的反应。发/pə/音时，神经放电开始与发声噗声（口腔呼出压力）之间的延迟，相比发/tə/和/kə/音时显著更短且变化更小，这表明该放电与/pə/发音的开始联系更为紧密。这些数据被解释为意味着眶下神经的单纤维对发/pə/音时口腔气压的升高做出反应。这种以及类似的感觉信息，可能被用于监测和控制发声所需气压的中枢神经机制。
