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细胞如何应对压力。

How cells respond to stress.

作者信息

Welch W J

机构信息

Lung Biology Center, University of California, San Francisco.

出版信息

Sci Am. 1993 May;268(5):56-64. doi: 10.1038/scientificamerican0593-56.

DOI:10.1038/scientificamerican0593-56
PMID:8097593
Abstract

During emergencies, cells produce stress proteins that repair damage. Inquiry into how they work offers promise for coping with infection, autoimmune disease and even cancer.

摘要

在紧急情况下,细胞会产生修复损伤的应激蛋白。探究这些蛋白的工作方式有望帮助应对感染、自身免疫性疾病甚至癌症。

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How cells respond to stress.细胞如何应对压力。
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Molecular chaperone functions of heat-shock proteins.热休克蛋白的分子伴侣功能
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[Heat shock pre-conditioning prevents disruption of actin cytoskeleton of endothelial cells due to ATP depletion: the role for heat shock protein HSP27].[热休克预处理可防止因ATP耗竭导致的内皮细胞肌动蛋白细胞骨架破坏:热休克蛋白HSP27的作用]
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Error-prone SOS repair can be error-free.易出错的SOS修复可以是无差错的。
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