粘附性F107菌毛和志贺样毒素II变异体（SLT-IIv）毒素在猪水肿病发病机制中的作用。
The role of adhesive F107 fimbriae and of SLT-IIv toxin in the pathogenesis of edema disease in pigs.
作者信息
Imberechts H, de Greve H, Hernalsteens J P, Schlicker C, Bouchet H, Pohl P, Charlier G, Bertschinger H U, Wild P, Vandekerckhove J
机构信息
Laboratory of Bacteriology, National Institute for Veterinary Research, Brussels, Belgium.
出版信息
Zentralbl Bakteriol. 1993 Apr;278(2-3):445-50. doi: 10.1016/s0934-8840(11)80861-3.
Colonization of the small intestine and the excretion of a toxin are important steps in the pathogenesis of edema disease in pigs. Although much is known about the chemical and biological characteristics of SLT-IIv toxin, its mode of action and its genetic determinant, F107 fimbriae were only recently described as colonization factors. Here we summarize our current knowledge about the virulence factors F107 fimbriae and SLT-IIv toxin.
小肠的定殖和毒素的排泄是猪水肿病发病机制中的重要步骤。尽管对SLT-IIv毒素的化学和生物学特性、其作用方式及其遗传决定因素已了解很多，但F107菌毛直到最近才被描述为定殖因子。在此，我们总结了目前关于毒力因子F107菌毛和SLT-IIv毒素的知识。
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