Seböková E, Klimes I, Moss R, Stolba P, Wiersma M M, Mitková A
Institute of Experimental Endocrinology, Slovak Academy of Sciences, Bratislava.
Ann N Y Acad Sci. 1993 Jun 14;683:218-27. doi: 10.1111/j.1749-6632.1993.tb35710.x.
Our data indicate that (a) the existence of a defect in the clearance of circulating TG and persistence of muscle TG deposition in the high sucrose-fed neonatal streptozotocin diabetic rat, which (b) can only be partially corrected by raised dietary n-3 PUFA intake. (c) Skeletal muscle of STZ type II-like diabetic rats contains about 40% less glucose transporters, and (d) this situation cannot be changed by any of the dietary treatments employed. (e) These findings indicate that muscle TG accumulation may have no direct relation to glucose transport in muscle.
（a）在高蔗糖喂养的新生链脲佐菌素糖尿病大鼠中，循环甘油三酯（TG）清除存在缺陷，肌肉TG沉积持续存在，且（b）提高膳食n-3多不饱和脂肪酸（PUFA）摄入量只能部分纠正这种情况。（c）链脲佐菌素诱导的II型糖尿病样大鼠的骨骼肌中葡萄糖转运蛋白含量约少40%，并且（d）采用的任何饮食治疗都无法改变这种情况。（e）这些发现表明，肌肉TG积累可能与肌肉中的葡萄糖转运没有直接关系。
