隔离及其问题：恶性疟原虫疟疾中受感染红细胞与内皮细胞的相互作用 - Suppr

Suppr

超能文献

Sequestration and its discontents: infected erythrocyte-endothelial cell interactions in Plasmodium falciparum malaria.

作者信息

Berendt A R

机构信息

Institute of Molecular Medicine, John Radcliffe Hospital, Headington, Oxford, UK.

出版信息

Res Immunol. 1993 Nov-Dec;144(9):740-5; discussion 754-62. doi: 10.1016/s0923-2494(93)80059-8.

DOI:10.1016/s0923-2494(93)80059-8

PMID:8159872

Abstract

摘要

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Sequestration and its discontents: infected erythrocyte-endothelial cell interactions in Plasmodium falciparum malaria.隔离及其问题：恶性疟原虫疟疾中受感染红细胞与内皮细胞的相互作用

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[Erythrocyte adhesion to the vascular endothelium].

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Plasmodium falciparum-infected erythrocytes bind ICAM-1 at a site distinct from LFA-1, Mac-1, and human rhinovirus.恶性疟原虫感染的红细胞在一个与淋巴细胞功能相关抗原-1、巨噬细胞抗原-1和人鼻病毒不同的位点结合细胞间黏附分子-1。

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Cytoadherence of Plasmodium falciparum-infected erythrocytes to microvascular endothelium is regulatable by cytokines and phorbol ester.恶性疟原虫感染的红细胞与微血管内皮细胞的细胞粘附可由细胞因子和佛波酯调节。

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Adhesion of sickle red cells to endothelium: myths and future directions.镰状红细胞与内皮细胞的黏附：误区与未来方向。

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Systemic endothelial activation occurs in both mild and severe malaria. Correlating dermal microvascular endothelial cell phenotype and soluble cell adhesion molecules with disease severity.全身性内皮细胞激活在轻度和重度疟疾中均会发生。将皮肤微血管内皮细胞表型和可溶性细胞黏附分子与疾病严重程度相关联。

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