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1
Adoptive transfer of polyclonal and cloned cytolytic T lymphocytes (CTL) specific for mouse AIDS-associated tumors is effective in preserving CTL responses: a measure of protection against LP-BM5 retrovirus-induced immunodeficiency.将针对小鼠艾滋病相关肿瘤的多克隆和克隆细胞毒性T淋巴细胞（CTL）进行过继转移，可有效维持CTL反应：这是一种抵御LP-BM5逆转录病毒诱导的免疫缺陷的保护措施。
J Virol. 1994 Jul;68(7):4679-84. doi: 10.1128/JVI.68.7.4679-4684.1994.
2
Cytotoxic T lymphocytes directed against MAIDS-associated tumors and cells from mice infected by the LP-BM5 MAIDS defective retrovirus.针对与MAIDS相关肿瘤以及感染LP - BM5 MAIDS缺陷逆转录病毒的小鼠细胞的细胞毒性T淋巴细胞。
Virology. 1994 Apr;200(1):292-6. doi: 10.1006/viro.1994.1189.
3
Anti-Gag cytolytic T lymphocytes specific for an alternative translational reading frame-derived epitope and resistance versus susceptibility to retrovirus-induced murine AIDS in F(1) mice.针对一个由替代翻译阅读框衍生的表位的抗Gag细胞溶解性T淋巴细胞以及F(1)小鼠对逆转录病毒诱导的鼠类艾滋病的抗性与易感性
Virology. 2000 Jul 5;272(2):438-49. doi: 10.1006/viro.2000.0339.
4
Cytolytic T lymphocytes specific for tumors and infected cells from mice with a retrovirus-induced immunodeficiency syndrome.针对患有逆转录病毒诱导的免疫缺陷综合征的小鼠的肿瘤细胞和受感染细胞的细胞毒性T淋巴细胞。
J Virol. 1992 May;66(5):3251-6. doi: 10.1128/JVI.66.5.3251-3256.1992.
5
Immunotherapy of murine retrovirus-induced acquired immunodeficiency by CD4 T regulatory cell depletion and PD-1 blockade.通过耗尽 CD4 T 调节性细胞和 PD-1 阻断治疗鼠逆转录病毒诱导的获得性免疫缺陷。
J Virol. 2011 Dec;85(24):13342-53. doi: 10.1128/JVI.00120-11. Epub 2011 Sep 14.
6
Evidence for a continued requirement for CD40/CD40 ligand (CD154) interactions in the progression of LP-BM5 retrovirus-induced murine AIDS.在LP - BM5逆转录病毒诱导的小鼠艾滋病进展过程中，持续需要CD40/CD40配体（CD154）相互作用的证据。
Virology. 1998 Feb 15;241(2):260-8. doi: 10.1006/viro.1997.8970.
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Characterization of the CD154-positive and CD40-positive cellular subsets required for pathogenesis in retrovirus-induced murine immunodeficiency.逆转录病毒诱导的小鼠免疫缺陷发病机制中所需的CD154阳性和CD40阳性细胞亚群的特征
J Virol. 2001 Apr;75(8):3581-9. doi: 10.1128/JVI.75.8.3581-3589.2001.
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Cytotoxic T lymphocytes to endogenous mouse retroviruses and mechanisms of retroviral escape.
Immunol Rev. 1999 Apr;168:271-86. doi: 10.1111/j.1600-065x.1999.tb01298.x.
9
Accelerated progression of a murine retrovirus-induced immunodeficiency syndrome in Fas mutant C57BL/6 lpr/lpr mice.Fas突变型C57BL/6 lpr/lpr小鼠中鼠逆转录病毒诱导的免疫缺陷综合征的加速进展
Microbiol Immunol. 1997;41(3):221-7. doi: 10.1111/j.1348-0421.1997.tb01194.x.
10
An alternative translational reading frame encodes an immunodominant retroviral CTL determinant expressed by an immunodeficiency-causing retrovirus.另一种翻译阅读框编码一种由导致免疫缺陷的逆转录病毒表达的免疫显性逆转录病毒CTL决定簇。
J Immunol. 1998 Jan 1;160(1):39-50.

引用本文的文献

1
Immunotherapy of murine retrovirus-induced acquired immunodeficiency by CD4 T regulatory cell depletion and PD-1 blockade.通过耗尽 CD4 T 调节性细胞和 PD-1 阻断治疗鼠逆转录病毒诱导的获得性免疫缺陷。
J Virol. 2011 Dec;85(24):13342-53. doi: 10.1128/JVI.00120-11. Epub 2011 Sep 14.
2
In vivo effects of locally secreted IL-10 on the murine antitumor immune response.局部分泌的白细胞介素-10对小鼠抗肿瘤免疫反应的体内效应。
Ann Surg Oncol. 1996 Jul;3(4):381-6. doi: 10.1007/BF02305668.
3
Antibody to the ligand for CD40 (gp39) inhibits murine AIDS-associated splenomegaly, hypergammaglobulinemia, and immunodeficiency in disease-susceptible C57BL/6 mice.针对CD40配体（gp39）的抗体可抑制疾病易感的C57BL/6小鼠中与艾滋病相关的脾肿大、高球蛋白血症和免疫缺陷。
J Virol. 1996 Apr;70(4):2569-75. doi: 10.1128/JVI.70.4.2569-2575.1996.

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Experiment and discussion on leukaemogenesis by cell-free extracts of radiation-induced leukaemia in mice.小鼠辐射诱导白血病无细胞提取物致白血病的实验与讨论
Int J Radiat Biol Relat Stud Phys Chem Med. 1962 Aug;5:339-44. doi: 10.1080/09553006214550911.
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GENETIC BASIS OF SUSCEPTIBILITY TO VIRAL LEUKAEMOGENESIS.病毒致白血病易感性的遗传基础。
Lancet. 1964 Dec 5;2(7371):1207-9. doi: 10.1016/s0140-6736(64)91043-8.
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An immunodominant Kb-restricted peptide from the p15E transmembrane protein of endogenous ecotropic murine leukemia virus (MuLV) AKR623 that restores susceptibility of a tumor line to anti-AKR/Gross MuLV cytotoxic T lymphocytes.一种来自内源性嗜亲性鼠白血病病毒（MuLV）AKR623的p15E跨膜蛋白的免疫显性Kb限制性肽，该肽可恢复肿瘤细胞系对抗AKR/格罗斯MuLV细胞毒性T淋巴细胞的敏感性。
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4
Cytotoxic T lymphocytes directed against MAIDS-associated tumors and cells from mice infected by the LP-BM5 MAIDS defective retrovirus.针对与MAIDS相关肿瘤以及感染LP - BM5 MAIDS缺陷逆转录病毒的小鼠细胞的细胞毒性T淋巴细胞。
Virology. 1994 Apr;200(1):292-6. doi: 10.1006/viro.1994.1189.
5
Characteristics of the cell populations involved in extra-thymic lymphosarcoma induced in C57BL/6 mice by RadLV-Rs.由RadLV-Rs诱导的C57BL/6小鼠胸腺外淋巴肉瘤中所涉及的细胞群体特征
Leuk Res. 1981;5(3):223-33. doi: 10.1016/0145-2126(81)90107-7.
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H-2D control of recovery from Friend virus leukemia: H-2D region influences the kinetics of the T lymphocyte response to Friend virus.H-2D对弗氏病毒白血病恢复的控制：H-2D区域影响T淋巴细胞对弗氏病毒反应的动力学。
J Exp Med. 1983 Jun 1;157(6):1736-45. doi: 10.1084/jem.157.6.1736.
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Immunopathology of B-cell lymphomas induced in C57BL/6 mice by dualtropic murine leukemia virus (MuLV).双嗜性鼠白血病病毒（MuLV）诱导C57BL/6小鼠发生B细胞淋巴瘤的免疫病理学
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The specificity of H-2-restricted cytotoxic T lymphocytes directed to AKR/Gross leukemia virus-induced tumors. II. Altered gp70 display and production of noninfectious virus particles by an insusceptible variant tumor.针对AKR/格罗斯白血病病毒诱导肿瘤的H-2限制性细胞毒性T淋巴细胞的特异性。II. 一种不易感的变异肿瘤中gp70展示的改变及非感染性病毒颗粒的产生
Eur J Immunol. 1983 Nov;13(11):871-7. doi: 10.1002/eji.1830131103.
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The specificity of H-2-restricted cytotoxic T lymphocytes directed to AKR/Gross leukemia virus-induced tumors. I. Isolation of a selectively resistant variant tumor subclone.针对AKR/格罗斯白血病病毒诱导肿瘤的H-2限制性细胞毒性T淋巴细胞的特异性。I. 一种选择性抗性变异肿瘤亚克隆的分离。
Eur J Immunol. 1983 Nov;13(11):863-70. doi: 10.1002/eji.1830131102.
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Specificity of cytolytic T cells directed against AKR/Gross virus-induced syngeneic leukemias: antibodies directed against H-2K, but not against viral proteins, inhibit lysis.针对AKR/格罗斯病毒诱导的同基因白血病的细胞溶解性T细胞的特异性：针对H-2K而非病毒蛋白的抗体可抑制细胞溶解。
J Immunol. 1980 Aug;125(2):647-55.

将针对小鼠艾滋病相关肿瘤的多克隆和克隆细胞毒性T淋巴细胞（CTL）进行过继转移，可有效维持CTL反应：这是一种抵御LP-BM5逆转录病毒诱导的免疫缺陷的保护措施。

Adoptive transfer of polyclonal and cloned cytolytic T lymphocytes (CTL) specific for mouse AIDS-associated tumors is effective in preserving CTL responses: a measure of protection against LP-BM5 retrovirus-induced immunodeficiency.

作者信息

Green W R, Green K A, Crassi K M

机构信息

Department of Microbiology, Dartmouth Medical School, Lebanon, New Hampshire 03756.

出版信息

J Virol. 1994 Jul;68(7):4679-84. doi: 10.1128/JVI.68.7.4679-4684.1994.

DOI:10.1128/JVI.68.7.4679-4684.1994
PMID:8207844
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC236398/
Abstract

Cytolytic T lymphocytes (CTL) can be raised against C57BL/6 B-cell lymphomas from mice with LP-BM5 murine leukemia virus-induced AIDS (MAIDS). Adoptive transfer of polyclonal anti-MAIDS tumor CTL or two CTL clones specific for the B6-1710 MAIDS lymphoma caused preservation of major histocompatibility complex-restricted and allogeneic CTL responses, which may be interpreted as indices of protection from LP-BM5 murine leukemia virus-induced immunodeficiency.

摘要

细胞溶解性T淋巴细胞（CTL）可以针对感染LP - BM5鼠白血病病毒诱发的获得性免疫缺陷综合征（MAIDS）小鼠的C57BL / 6 B细胞淋巴瘤产生。多克隆抗MAIDS肿瘤CTL或针对B6 - 1710 MAIDS淋巴瘤的两个CTL克隆的过继转移导致主要组织相容性复合体限制的和同种异体CTL反应得以保留，这可以被解释为免受LP - BM5鼠白血病病毒诱发免疫缺陷的指标。