Shimasaki T, Oshikiri T, Uchino H, Washio M
Second Department of Surgery, Yamagata University School of Medicine, Japan.
Nihon Kyobu Geka Gakkai Zasshi. 1993 Oct;41(10):2029-33.
It is generally recognized that if skeletal muscle is stimulated corresponding with the heart rate to assist the cardiac performance, the contraction power of the skeletal muscle decreases in a few minutes. It becomes necessary that the skeletal muscle is electrically stimulated in 6-9 weeks to lead into the transformation or that the muscle is laid without working after operation in 6-9 weeks to wait for growing of the collateral vessels. The observation time of the fatigue phenomenon in many studies is, however, short and it is not understandable that the contraction power decreases near to zero although the type I fibers remain. So we studied the relationship between the time and the pressure assist of the skeletal muscle ventricle (SMV). It was concluded that 1) the cause of the power decrease is not only the fatigue but the operative invasion. 2) in our setting, when SMV was stimulated in the ratio of 1:4 or 1:10 to the heart rate, the power recovered within 3 hours. 3) if the initial setting of the drive is light enough, the preconditioning or the vascular delay will be unnecessary and the working transformation or the in situ training will practicable.
人们普遍认识到，如果骨骼肌与心率同步受到刺激以辅助心脏功能，骨骼肌的收缩力会在几分钟内下降。因此有必要在6至9周内对骨骼肌进行电刺激以促成其转变，或者在术后6至9周内让肌肉处于不工作状态以等待侧支血管生长。然而，许多研究中对疲劳现象的观察时间较短，而且尽管I型纤维仍然存在，但收缩力降至接近零的情况却难以理解。所以我们研究了时间与骨骼肌心室（SMV）压力辅助之间的关系。得出的结论是：1）力量下降的原因不仅是疲劳，还包括手术侵袭。2）在我们的实验设置中，当SMV以1:4或1:10的比例与心率同步刺激时，力量在3小时内恢复。3）如果驱动的初始设置足够轻，就无需进行预处理或血管延迟，工作转变或原位训练将切实可行。
