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系统性红斑狼疮患者血浆置换诱发的代谢性碱中毒。

Metabolic alkalosis induced by plasmapheresis in a patient with systemic lupus erythematosus.

作者信息

Choi M Y, Lee J D, Lee S H, Park I S, Woo J Y, Choi E J, Chang Y S, Bang B K

机构信息

Department of Internal Medicine, Catholic University Medical College, Seoul, Korea.

出版信息

J Korean Med Sci. 1993 Jun;8(3):207-9. doi: 10.3346/jkms.1993.8.3.207.

DOI:10.3346/jkms.1993.8.3.207
PMID:8240751
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3053737/
Abstract

We report a patient with systemic lupus erythematosus (SLE), who had developed metabolic alkalosis during plasmapheresis. The metabolic alkalosis could be promptly corrected by reducing the amount of citrate load. The development of metabolic alkalosis can be explained by the citrate load during plasmapheresis. Careful monitoring of acid base status is mandatory in patients with limited renal function and the reduction of citrate load may be advisable in plasmapheresis.

摘要

我们报告了一名系统性红斑狼疮(SLE)患者,该患者在血浆置换过程中出现了代谢性碱中毒。通过减少柠檬酸盐负荷量,代谢性碱中毒可迅速得到纠正。代谢性碱中毒的发生可通过血浆置换过程中的柠檬酸盐负荷来解释。对于肾功能有限的患者,必须仔细监测酸碱状态,在血浆置换中减少柠檬酸盐负荷量可能是可取的。

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Metabolic alkalosis induced by plasmapheresis in a patient with systemic lupus erythematosus.系统性红斑狼疮患者血浆置换诱发的代谢性碱中毒。
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