Nussselt L, Kockott G
EEG EMG Z Elektroenzephalogr Elektromyogr Verwandte Geb. 1976 Mar;7(1):42-8.
The study's goal was the examination of the hypothesis that transsexualism develops on a diminution of sexual drive which itself is a result of temporal lobe dysfunction. 28 transsexuals were examined. More than 30% of this group showed pathologic findings in EEG recordings. Most of the pathological findings were situated in the temporal region. Four transsexuals with pathologic EEG findings mentioned a brain injury in their personal history. In all but one case this injury had taken place after manifestation of transsexual behavior. There was no correlation between depressed sexual drive and pathologic electroencephalographic changes. Generally genetic male transsexuals showed a diminuation of sexual drive only by taking estradiol. This is interpreted as a hypogonadotropic hypogonadism. The electroencephalographic findings in 9 or 28 cases of transsexualism with an increase of epsilon-waves mostly in the temporal region are discussed in the context of single case studies and regarded as a reference to temporal lobe dysfunction. This dysfunction is interpreted as a biologic factor in the etiology of transsexualism.
该研究的目的是检验一种假说，即易性癖的形成是由于性欲减退，而性欲减退本身是颞叶功能障碍的结果。对28名易性癖者进行了检查。该组中超过30%的人在脑电图记录中显示出病理结果。大多数病理结果位于颞叶区域。4名脑电图有病理结果的易性癖者在个人病史中提到有脑损伤。除1例之外，所有这些损伤均发生在易性癖行为出现之后。性欲减退与脑电图的病理变化之间没有相关性。一般来说，遗传男性易性癖者只有在服用雌二醇后才会出现性欲减退。这被解释为低促性腺激素性性腺功能减退。结合单病例研究对28例易性癖中9例脑电图结果进行了讨论，这些结果大多在颞叶区域出现ε波增多，被视为与颞叶功能障碍有关。这种功能障碍被解释为易性癖病因中的一个生物学因素。
