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Association of hyperphospholipasemia A2 with multiple system organ dysfunction due to salicylate intoxication.

作者信息

Vadas P, Schouten B D, Stefanski E, Scott K, Pruzanski W

机构信息

Wellesley Hospital Research Institute, University of Toronto, ON, Canada.

出版信息

Crit Care Med. 1993 Jul;21(7):1087-91. doi: 10.1097/00003246-199307000-00027.

DOI:10.1097/00003246-199307000-00027
PMID:8319465
Abstract
摘要

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Association of hyperphospholipasemia A2 with multiple system organ dysfunction due to salicylate intoxication.高磷脂酶A2与水杨酸盐中毒所致多系统器官功能障碍的关联。
Crit Care Med. 1993 Jul;21(7):1087-91. doi: 10.1097/00003246-199307000-00027.
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引用本文的文献

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Inhibition of the activity of pro-inflammatory secretory phospholipase A(2) by acute phase proteins.急性期蛋白对促炎分泌型磷脂酶 A(2)活性的抑制作用。
Mediators Inflamm. 1996;5(3):196-201. doi: 10.1155/S0962935196000270.
2
Tenidap sodium inhibits secretory non-pancreatic phospholipase A(2) synthesis by foetal rat calvarial osteoblasts.替地那普利钠抑制胎鼠颅骨成骨细胞分泌型非胰腺磷脂酶 A2 的合成。
Mediators Inflamm. 1995;4(1):67-70. doi: 10.1155/S0962935195000123.
3
Induction of secretory phospholipase A2 confirms the systemic inflammatory nature of adjuvant arthritis.
分泌型磷脂酶A2的诱导证实了佐剂性关节炎的全身炎症性质。
Inflammation. 1998 Apr;22(2):161-73. doi: 10.1023/a:1022336006109.
4
Therapy with interleukin-2 induces the systemic release of phospholipase-A2.白细胞介素-2治疗可诱导磷脂酶A2的全身释放。
Cancer Immunol Immunother. 1995 Nov;41(5):287-92. doi: 10.1007/BF01517216.
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Serum amyloid A protein enhances the activity of secretory non-pancreatic phospholipase A2.血清淀粉样蛋白A可增强分泌型非胰腺磷脂酶A2的活性。
Biochem J. 1995 Jul 15;309 ( Pt 2)(Pt 2):461-4. doi: 10.1042/bj3090461.