Murine models for study of lipoprotein metabolism and atherosclerosis.
作者信息
Witztum J L
出版信息
J Clin Invest. 1993 Aug;92(2):536-7. doi: 10.1172/JCI116619.
Abstract
摘要
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Murine models for study of lipoprotein metabolism and atherosclerosis.用于研究脂蛋白代谢和动脉粥样硬化的小鼠模型。
J Clin Invest. 1993 Aug;92(2):536-7. doi: 10.1172/JCI116619.
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[Lipoprotein metabolism and atherosclerosis--experimental principles].[脂蛋白代谢与动脉粥样硬化——实验原理]
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Hepatic expression of apolipoprotein E inhibits progression of atherosclerosis without reducing cholesterol levels in LDL receptor-deficient mice.载脂蛋白E在肝脏中的表达可抑制动脉粥样硬化的进展，而不会降低低密度脂蛋白受体缺陷小鼠的胆固醇水平。
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Increased low-density lipoprotein oxidation and impaired high-density lipoprotein antioxidant defense are associated with increased macrophage homing and atherosclerosis in dyslipidemic obese mice: LCAT gene transfer decreases atherosclerosis.在血脂异常的肥胖小鼠中，低密度脂蛋白氧化增加和高密度脂蛋白抗氧化防御受损与巨噬细胞归巢增加及动脉粥样硬化相关：卵磷脂胆固醇酰基转移酶基因转移可减轻动脉粥样硬化。
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Human apolipoprotein AI mimetic peptides for the treatment of atherosclerosis.用于治疗动脉粥样硬化的人载脂蛋白AI模拟肽。
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Upregulation of selective cholesteryl ester uptake pathway in mice with deletion of low-density lipoprotein receptor function.低密度脂蛋白受体功能缺失小鼠中选择性胆固醇酯摄取途径的上调。
J Cell Physiol. 1999 Aug;180(2):190-202. doi: 10.1002/(SICI)1097-4652(199908)180:2<190::AID-JCP7>3.0.CO;2-Z.
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Important role for bone marrow-derived cholesteryl ester transfer protein in lipoprotein cholesterol redistribution and atherosclerotic lesion development in LDL receptor knockout mice.骨髓源性胆固醇酯转运蛋白在低密度脂蛋白受体敲除小鼠的脂蛋白胆固醇再分布及动脉粥样硬化病变发展中起重要作用。
Circ Res. 2007 Mar 16;100(5):678-85. doi: 10.1161/01.RES.0000260202.79927.4f. Epub 2007 Feb 9.
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The proper study of mankind.对人类的恰当研究。
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Hypercholesterolemia is associated with a T helper (Th) 1/Th2 switch of the autoimmune response in atherosclerotic apo E-knockout mice.高胆固醇血症与动脉粥样硬化载脂蛋白E基因敲除小鼠自身免疫反应中的辅助性T细胞（Th）1/Th2转换有关。
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本文引用的文献
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Adenovirus-mediated transfer of low density lipoprotein receptor gene acutely accelerates cholesterol clearance in normal mice.腺病毒介导的低密度脂蛋白受体基因转移可急性加速正常小鼠的胆固醇清除。
Proc Natl Acad Sci U S A. 1993 Apr 1;90(7):2812-6. doi: 10.1073/pnas.90.7.2812.
2
Hypercholesterolemia in low density lipoprotein receptor knockout mice and its reversal by adenovirus-mediated gene delivery.低密度脂蛋白受体基因敲除小鼠的高胆固醇血症及其通过腺病毒介导的基因递送的逆转。
J Clin Invest. 1993 Aug;92(2):883-93. doi: 10.1172/JCI116663.
3
Variation in susceptibility to atherosclerosis among inbred strains of mice.近交系小鼠对动脉粥样硬化易感性的差异。
Atherosclerosis. 1985 Oct;57(1):65-73. doi: 10.1016/0021-9150(85)90138-8.
4
Deletion in the gene for the low-density-lipoprotein receptor in a majority of French Canadians with familial hypercholesterolemia.在大多数患有家族性高胆固醇血症的法裔加拿大人中，低密度脂蛋白受体基因存在缺失。
N Engl J Med. 1987 Sep 17;317(12):734-7. doi: 10.1056/NEJM198709173171204.
5
Ath-1, a gene determining atherosclerosis susceptibility and high density lipoprotein levels in mice.Ath-1，一种决定小鼠动脉粥样硬化易感性和高密度脂蛋白水平的基因。
Proc Natl Acad Sci U S A. 1987 Jun;84(11):3763-7. doi: 10.1073/pnas.84.11.3763.
6
Ath-2, a second gene determining atherosclerosis susceptibility and high density lipoprotein levels in mice.Ath-2，小鼠中决定动脉粥样硬化易感性和高密度脂蛋白水平的第二个基因。
Genetics. 1989 May;122(1):163-8. doi: 10.1093/genetics/122.1.163.
7
Lipoproteins and atherogenesis. Current concepts.
JAMA. 1990 Dec 19;264(23):3047-52.
8
Inhibition of early atherogenesis in transgenic mice by human apolipoprotein AI.人载脂蛋白AI对转基因小鼠早期动脉粥样硬化形成的抑制作用。
Nature. 1991 Sep 19;353(6341):265-7. doi: 10.1038/353265a0.
9
Effects of atherogenic diet consumption on lipoproteins in mouse strains C57BL/6 and C3H.致动脉粥样化饮食摄入对C57BL/6和C3H小鼠品系脂蛋白的影响。
J Lipid Res. 1991 Apr;32(4):559-68.
10
Role of oxidized low density lipoprotein in atherogenesis.氧化型低密度脂蛋白在动脉粥样硬化形成中的作用。
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