Niggli H J
Cosmital SA (Research Company of Wella AG, Darmstadt) Germany.
J Photochem Photobiol B. 1993 May;18(2-3):281-5. doi: 10.1016/1011-1344(93)80076-l.
Photons participate in many atomic and molecular interactions and changes in the physical universe. In recent years sophisticated detection procedures for the measurement of ultraweak photons in a variety of different cells have been performed leading to the conclusion that plant, animal and human cells emit ultraweak photons. Using an extremely low-noise, high-sensitive photon-counting system, which allows maximal exploitation of the potential capabilities of a photomultiplier tube, ultraweak photons were quantitated in human skin fibroblasts. It was found that light from an artificial sunlight source induces ultraweak photon emission in these cells. However, the results demonstrate that this induction is significantly lower in normal fibroblasts compared with those obtained from a donor suffering from xeroderma pigmentosum disease group A, a disease characterized by deficient repair of DNA. The largest increase in ultraweak photon emission after UV exposure was measured in mitomycin-C-induced post-mitotic xeroderma pigmentosum cells which showed 10-20 times higher ultraweak photon intensities than mitotic UV-irradiated normal cells. These data suggest that xeroderma pigmentosum cells tend to lose the capacity of efficient storage of ultraweak photons, indicating the existence of an efficient intracellular photon trapping system within human cells.
光子参与了物理宇宙中的许多原子和分子相互作用及变化。近年来，已经开展了复杂的检测程序来测量各种不同细胞中的超微弱光子，从而得出植物、动物和人类细胞都会发射超微弱光子的结论。使用一种极低噪声、高灵敏度的光子计数系统，该系统能最大程度地发挥光电倍增管的潜在能力，对人类皮肤成纤维细胞中的超微弱光子进行了定量。研究发现，来自人工太阳光源的光会诱导这些细胞发射超微弱光子。然而，结果表明，与从患有A型色素性干皮病的供体获得的成纤维细胞相比，正常成纤维细胞中的这种诱导作用明显更低，A型色素性干皮病是以DNA修复缺陷为特征的一种疾病。在丝裂霉素C诱导的有丝分裂后色素性干皮病细胞中，测量到紫外线照射后超微弱光子发射的最大增加，这些细胞显示出的超微弱光子强度比有丝分裂期紫外线照射的正常细胞高10到20倍。这些数据表明，色素性干皮病细胞往往会失去有效储存超微弱光子的能力，这表明人类细胞内存在一个有效的细胞内光子捕获系统。
