• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

相似文献

1
Changes in complement and immunoglobulin G receptor expression on neutrophils associated with Porphyromonas gingivalis-induced inhibition of phagocytosis.牙龈卟啉单胞菌诱导的中性粒细胞吞噬作用抑制与补体及免疫球蛋白G受体表达的变化
Infect Immun. 1993 Aug;61(8):3533-5. doi: 10.1128/iai.61.8.3533-3535.1993.
2
Differential expression of CR3, Fc epsilon RII and Fc gamma RIII on polymorphonuclear leukocytes in gingival crevicular fluid.龈沟液中多形核白细胞上CR3、FcεRII和FcγRIII的差异表达
J Periodontal Res. 1993 Sep;28(5):363-72. doi: 10.1111/j.1600-0765.1993.tb01080.x.
3
Activation of polymorphonuclear neutrophilic granulocytes following burn injury: alteration of Fc-receptor and complement-receptor expression and of opsonophagocytosis.烧伤后多形核中性粒细胞的激活：Fc受体、补体受体表达及调理吞噬作用的改变
J Trauma. 1994 Feb;36(2):161-7. doi: 10.1097/00005373-199402000-00001.
4
Neutrophils from preterm neonates and adults show similar cell surface receptor expression: analysis using a whole blood assay.
Biol Neonate. 1995;67(1):26-33. doi: 10.1159/000244139.
5
Inhibition of C3 and IgG proteolysis enhances phagocytosis of Porphyromonas gingivalis.抑制C3和IgG蛋白水解可增强牙龈卟啉单胞菌的吞噬作用。
J Immunol. 1993 Dec 15;151(12):7016-29.
6
Effective in vitro clearance of Porphyromonas gingivalis by Fc alpha receptor I (CD89) on gingival crevicular neutrophils.牙龈沟中性粒细胞上的Fcα受体I（CD89）对牙龈卟啉单胞菌具有有效的体外清除作用。
Infect Immun. 2001 May;69(5):2935-42. doi: 10.1128/IAI.69.5.2935-2942.2001.
7
Effects of Porphyromonas gingivalis culture products on human polymorphonuclear leukocyte function.牙龈卟啉单胞菌培养产物对人多形核白细胞功能的影响。
FEMS Microbiol Immunol. 1990 Dec;2(5-6):285-93. doi: 10.1111/j.1574-6968.1990.tb03531.x.
8
Loss of Fc gamma receptor and impaired phagocytosis of polymorphonuclear leukocytes in gingival crevicular fluid.龈沟液中Fcγ受体缺失及多形核白细胞吞噬功能受损。
J Periodontal Res. 1997 Jul;32(5):439-46. doi: 10.1111/j.1600-0765.1997.tb00556.x.
9
CR3 (Mac-1, alpha M beta 2, CD11b/CD18) and Fc gamma RIII cooperate in generation of a neutrophil respiratory burst: requirement for Fc gamma RIII and tyrosine phosphorylation.CR3（巨噬细胞-1抗原，αMβ2，CD11b/CD18）与FcγRIII协同作用产生中性粒细胞呼吸爆发：对FcγRIII和酪氨酸磷酸化的需求
J Cell Biol. 1994 Jun;125(6):1407-16. doi: 10.1083/jcb.125.6.1407.
10
Assessment of certain neutrophil receptors, opsonophagocytosis and soluble intercellular adhesion molecule-1 (ICAM-1) following thermal injury.热损伤后某些中性粒细胞受体、调理吞噬作用及可溶性细胞间黏附分子-1（ICAM-1）的评估。
Burns. 1999 Aug;25(5):395-401. doi: 10.1016/s0305-4179(98)00164-8.

引用本文的文献

1
Phagocytic activity of peripheral blood and crevicular phagocytes in health and periodontal disease.健康与牙周疾病状态下外周血及龈沟吞噬细胞的吞噬活性
J Indian Soc Periodontol. 2010 Jan;14(1):8-11. doi: 10.4103/0972-124X.65427.
2
Effective in vitro clearance of Porphyromonas gingivalis by Fc alpha receptor I (CD89) on gingival crevicular neutrophils.牙龈沟中性粒细胞上的Fcα受体I（CD89）对牙龈卟啉单胞菌具有有效的体外清除作用。
Infect Immun. 2001 May;69(5):2935-42. doi: 10.1128/IAI.69.5.2935-2942.2001.
3
Porphyromonas gingivalis infection of oral epithelium inhibits neutrophil transepithelial migration.牙龈卟啉单胞菌感染口腔上皮会抑制中性粒细胞经上皮迁移。
Infect Immun. 1997 Oct;65(10):3983-90. doi: 10.1128/iai.65.10.3983-3990.1997.

本文引用的文献

1
Protein measurement with the Folin phenol reagent.使用福林酚试剂进行蛋白质测定。
J Biol Chem. 1951 Nov;193(1):265-75.
2
Neutrophil chemotaxis dysfunction in human periodontitis.人类牙周炎中的中性粒细胞趋化功能障碍
Infect Immun. 1980 Jan;27(1):124-32. doi: 10.1128/iai.27.1.124-132.1980.
3
Phagocytosis: flow cytometric quantitation with fluorescent microspheres.吞噬作用：使用荧光微球进行流式细胞术定量分析。
Science. 1982 Jan 1;215(4528):64-6. doi: 10.1126/science.7053559.
4
Degradation of immunoglobulins A2, A2, and G by suspected principal periodontal pathogens.疑似主要牙周病原体对免疫球蛋白A2、A2和G的降解作用。
Infect Immun. 1981 Dec;34(3):757-65. doi: 10.1128/iai.34.3.757-765.1981.
5
Quantitation of neutrophil phagocytosis, using fluorescent latex beads. Correlation of microscopy and flow cytometry.使用荧光乳胶微球对中性粒细胞吞噬作用进行定量分析。显微镜检查与流式细胞术的相关性。
J Lab Clin Med. 1981 Sep;98(3):374-81.
6
Inhibition of neutrophil chemotaxis by soluble bacterial products.可溶性细菌产物对中性粒细胞趋化作用的抑制
J Periodontol. 1982 Aug;53(8):502-8. doi: 10.1902/jop.1982.53.8.502.
7
Neutrophil receptor modulation in the pathogenesis of periodontal diseases.中性粒细胞受体调节在牙周疾病发病机制中的作用
J Dent Res. 1984 Mar;63(3):452-4. doi: 10.1177/00220345840630031701.
8
Human neutrophils increase expression of C3bi as well as C3b receptors upon activation.人类中性粒细胞在激活后会增加C3bi以及C3b受体的表达。
J Clin Invest. 1984 Nov;74(5):1566-71. doi: 10.1172/JCI111572.
9
Characterization of a trypsin-like protease from the bacterium Bacteroides gingivalis isolated from human dental plaque.从人类牙菌斑中分离出的牙龈拟杆菌中一种类胰蛋白酶的特性研究
Arch Oral Biol. 1984;29(7):559-64. doi: 10.1016/0003-9969(84)90078-5.
10
Purification and characterization of a protease from Bacteroides gingivalis 381.牙龈卟啉单胞菌381蛋白酶的纯化与特性分析
Infect Immun. 1987 Feb;55(2):420-7. doi: 10.1128/iai.55.2.420-427.1987.

牙龈卟啉单胞菌诱导的中性粒细胞吞噬作用抑制与补体及免疫球蛋白G受体表达的变化

Changes in complement and immunoglobulin G receptor expression on neutrophils associated with Porphyromonas gingivalis-induced inhibition of phagocytosis.

作者信息

Tai H, Kobayashi T, Hara K

机构信息

Department of Periodontology, Niigata University School of Dentistry, Japan.

出版信息

Infect Immun. 1993 Aug;61(8):3533-5. doi: 10.1128/iai.61.8.3533-3535.1993.

DOI:10.1128/iai.61.8.3533-3535.1993
PMID:8392973
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC281034/
Abstract

We examined the phagocytic capacity and receptor expression on neutrophils stimulated with Porphyromonas gingivalis soluble products. Stimulated neutrophils had decreased phagocytic capacities and altered expression of CR1, CR3, Fc gamma RII, and Fc gamma RIII. For cases in which TLCK (N-alpha-p-tosyl-L-lysine chloromethyl ketone) neutralized the effects of the stimuli, the P. gingivalis-derived factors causing the phenomena seem to be trypsin-like proteases.

摘要

我们检测了牙龈卟啉单胞菌可溶性产物刺激的中性粒细胞的吞噬能力和受体表达。受刺激的中性粒细胞吞噬能力下降，补体受体1（CR1）、补体受体3（CR3）、FcγRII和FcγRIII的表达发生改变。对于N-α-对甲苯磺酰-L-赖氨酸氯甲基酮（TLCK）中和刺激作用的情况，引起这些现象的牙龈卟啉单胞菌衍生因子似乎是类胰蛋白酶。