Rama B N, Varghese J, Genova G, Kumar S
Creighton Cardiac Center, Omaha, NE 68131-2044.
Nebr Med J. 1993 Jun;78(6):151-4.
We have presented the case of a 34 year old male patient who was admitted with severe metabolic alkalosis (MA). Peak serum HCO3 was 96 mg/dl and compensatory PCO2 was 95 which, to our knowledge, has never been reported before in a patient with MA. MA was probably generated by consumption of high amount of NaHCO3 and renal impairment and maintained by impaired renal function due to volume depletion hypokalemia and hyochloremia. The patient was successfully treated with IV administration of saline and KCL.
我们报告了一名34岁男性患者的病例，该患者因严重代谢性碱中毒（MA）入院。血清碳酸氢根（HCO3）峰值为96mg/dl，代偿性二氧化碳分压（PCO2）为95，据我们所知，此前从未有MA患者出现过这种情况。MA可能是由于大量摄入碳酸氢钠和肾功能损害所致，并因容量耗竭、低钾血症和低氯血症导致的肾功能受损而持续存在。该患者通过静脉输注生理盐水和氯化钾成功治愈。
