前列腺素作为炎症的调节剂而非介质。 - Suppr

Suppr

超能文献

Prostaglandins as modulators rather than mediators of inflammation.

作者信息

Weissmann G

机构信息

Division of Rheumatology, New York University Medical Center, NY.

出版信息

J Lipid Mediat. 1993 Mar-Apr;6(1-3):275-86.

PMID:8395250

Abstract

摘要

相似文献

Prostaglandins as modulators rather than mediators of inflammation.前列腺素作为炎症的调节剂而非介质。

J Lipid Mediat. 1993 Mar-Apr;6(1-3):275-86.

[Biochemical, pharmacological and therapeutic aspects of skin metabolism of arachidonic acid].

Ann Dermatol Venereol. 1986;113(12):1289-93.

Prostaglandins and inflammation: receptor/cyclase coupling as an explanation of why PGEs and PGI2 inhibit functions of inflammatory cells.前列腺素与炎症：受体/环化酶偶联作为PGEs和PGI2抑制炎症细胞功能原因的解释

Adv Prostaglandin Thromboxane Res. 1980;8:1637-53.

Eicosanoids: lipid mediators of inflammation in transplantation.类二十烷酸：移植炎症中的脂质介质

Springer Semin Immunopathol. 2003 Sep;25(2):215-27. doi: 10.1007/s00281-003-0132-4.

Inflammation, prostaglandins, and loss of function.炎症、前列腺素与功能丧失。

J Lipid Mediat. 1993 Mar-Apr;6(1-3):287-93.

Prostaglandins and inflammation: the cyclooxygenase controversy.前列腺素与炎症：环氧化酶之争

Arch Immunol Ther Exp (Warsz). 2000;48(6):473-80.

Autacoids and some mediators in allergic and non allergic forms.过敏和非过敏形式中的自体活性物质及一些介质。

Boll Ist Sieroter Milan. 1986;65(3):169-92.

Prostaglandins and leukotrienes.前列腺素和白三烯。

Metab Pediatr Syst Ophthalmol (1985). 1985;8(4):148-9.

[Effect of inflammation mediators and antirheumatic drugs on the production of superoxide anion (O2-) by polymorphonuclear cells in normals and rheumatoid arthritis].[炎症介质和抗风湿药物对正常人和类风湿关节炎患者多形核细胞超氧阴离子（O2-）产生的影响]

Boll Soc Ital Biol Sper. 1982 Apr 15;58(7):392-6.

Immunology. Telling the brain about pain.免疫学。向大脑传递疼痛信息。

Nature. 2001 Mar 22;410(6827):425, 427. doi: 10.1038/35068663.

引用本文的文献

Severe hematopoietic stem cell inflammation compromises chronic granulomatous disease gene therapy.严重的造血干细胞炎症会损害慢性肉芽肿病的基因治疗。

Cell Rep Med. 2023 Feb 21;4(2):100919. doi: 10.1016/j.xcrm.2023.100919. Epub 2023 Jan 26.

NSAIDs-dependent adaption of the mitochondria-proteasome system in immortalized human cardiomyocytes.依赖 NSAIDs 的人永生化心肌细胞中线粒体-蛋白酶体系统的适应性改变。

Sci Rep. 2020 Oct 27;10(1):18337. doi: 10.1038/s41598-020-75394-x.

The effect of ozone exposure on the release of eicosanoids in guinea-pig BAL fluid in relation to cellular damage and inflammation.臭氧暴露对豚鼠 BAL 液中花生四烯酸释放的影响与细胞损伤和炎症的关系。

Mediators Inflamm. 1997;6(5-6):355-61. doi: 10.1080/09629359791497.

Prostanoids as friends, not foes: further evidence from the interference by cycloxygenase-inhibitory drugs when inducing tolerance to experimental arthritigens in rats.前列腺素是友非敌：环氧化酶抑制药物在诱导大鼠对实验性关节炎抗原产生耐受性时的干扰作用提供的进一步证据

Inflammopharmacology. 2005;12(5-6):481-92. doi: 10.1163/156856005774382788.

Early vascular permeability in murine experimental peritonitis is co-mediated by resident peritoneal macrophages and mast cells: crucial involvement of macrophage-derived cysteinyl-leukotrienes.在小鼠实验性腹膜炎中，早期血管通透性由驻留腹膜巨噬细胞和肥大细胞共同介导：巨噬细胞衍生的半胱氨酰白三烯起关键作用。

Inflammation. 2002 Apr;26(2):61-71. doi: 10.1023/a:1014837110735.

Prognostic factors associated with improved outcome of Escherichia coli bacteremia in a Finnish university hospital.

Eur J Clin Microbiol Infect Dis. 1997 Feb;16(2):125-34. doi: 10.1007/BF01709471.

Prostaglandin E2 requirement for transforming growth factor beta 1 inhibition of elicited macrophage 14 kDa phospholipase A2 release.前列腺素E2对转化生长因子β1抑制诱导型巨噬细胞14 kDa磷脂酶A2释放的必要性。

Br J Pharmacol. 1995 Nov;116(6):2575-81. doi: 10.1111/j.1476-5381.1995.tb17210.x.

Inducible isoforms of cyclooxygenase and nitric-oxide synthase in inflammation.炎症中环氧合酶和一氧化氮合酶的诱导型同工酶

Proc Natl Acad Sci U S A. 1994 Mar 15;91(6):2046-50. doi: 10.1073/pnas.91.6.2046.

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验