Kunath U
Langenbecks Arch Chir. 1977;343(2):161-72. doi: 10.1007/BF01262007.
For the first time, the capillary space-bursa infracardiaca-which is present in human evolution, is investigated and described in reference to the pathogenesis of sliding hiatal hernia. This bursa at the right dorsal wall of the esophagus can obliterate, persist or even communicate with the bursa omentalis. It supports the cranially sliding movement of the gastroesophageal junction and reduces the slope posture of the plane of esophageal entry into the stomach. Extensive sliding movement permits the fat of omentum minus to develop a lipoma between the bursa and esophageal wall. With regard to the known specific muscle architecture of esophagus (Stelzner, 1968) the upward tilting of the plane of entry is followed by the opening of the cardia and by reflux. The bursa enlarges the postural change of the plane of entry. It can be considered as a disposing factor in the development of sliding hiatal hernia. This is evaluated by a lot of observations and with regard to former experiments.
首次对人类进化过程中出现的毛细血管间隙-心包下囊进行了研究,并参照滑动性食管裂孔疝的发病机制对其进行了描述。食管右后壁的这个囊可以闭塞、持续存在,甚至与网膜囊相通。它支持胃食管交界处的向上滑动运动,并减小食管进入胃的平面的倾斜姿势。广泛的滑动运动会使小网膜脂肪在囊和食管壁之间形成脂肪瘤。关于已知的食管特定肌肉结构(施特尔兹纳,1968年),入口平面向上倾斜后会导致贲门打开并出现反流。该囊扩大了入口平面的姿势变化。它可被视为滑动性食管裂孔疝发生发展的一个诱发因素。这是通过大量观察以及参考以前的实验评估得出的。