Kilgour R D, Gariépy P, Rehel R
Department of Exercise Science, Concordia University, Montreal, Quebec, Canada.
Aviat Space Environ Med. 1993 Mar;64(3 Pt 1):224-9.
Although the central cardiovascular adjustments to exercise in the heat have been identified, little is known about the post-exercise hemodynamics during recovery from exercise and heat stress. This study examined heart rate (HR), stroke index (SI), cardiac index (CI), systemic vascular resistance (SVR), systolic (SBP) and diastolic (DBP) blood pressure in 8 males during 15 min of passive seated recovery preceded by 30 min of cycle ergometry (60% VO2max) on two separate occasions: under control (C) and heat stress (HS) conditions. During both recovery conditions, SI significantly declined (p < 0.05) to below pre-exercise values. No differences were observed between groups with respect to SI. The decrease in recovery HR was slower (p < 0.05) in HS than C. The greater elevation in HR during HS accounted for the relative increase in CI above that observed prior to exercise. The estimated SVR measured immediately following exercise in both groups was lower (p < 0.05) than pre-exercise values. By 5 min of C recovery, SVR returned to baseline values but remained significantly depressed (p < 0.05) for the entire HS condition. These results indicate that the pressor responses were attenuated during HS; however, CI was maintained above pre-exercise levels due to higher HR responses compensating for the reduction in SI. Stimulation of the baroreceptor reflex and increased myocardial contractility could possibly explain the maintenance of output at a time when preload and afterload were reduced.
尽管已经明确了热环境下运动时的中枢心血管调节机制,但对于运动及热应激恢复过程中的运动后血液动力学却知之甚少。本研究在两种不同条件下(对照条件(C)和热应激条件(HS)),对8名男性进行了研究。先进行30分钟的蹬车运动(60%最大摄氧量),之后进行15分钟的被动坐姿恢复,期间测量心率(HR)、每搏指数(SI)、心指数(CI)、全身血管阻力(SVR)、收缩压(SBP)和舒张压(DBP)。在两种恢复条件下,SI均显著下降(p<0.05)至运动前值以下。两组之间在SI方面未观察到差异。HS组恢复过程中心率下降较慢(p<0.05)。HS期间心率升高幅度较大,这导致CI相对于运动前观察值有所增加。两组运动后立即测得的估计SVR均低于运动前值(p<0.05)。在C恢复5分钟时,SVR恢复到基线值,但在整个HS条件下仍显著低于基线(p<0.05)。这些结果表明,在HS期间升压反应减弱;然而,由于较高的心率反应补偿了SI的降低,CI维持在运动前水平以上。压力感受器反射的刺激和心肌收缩力的增加可能解释了在预负荷和后负荷降低时心输出量的维持。